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Search term: adipocyte

Results 1 - 100 of 135 > >>
EC Number
Source Tissue
Commentary
Reference
11beta-hydroxysteroid dehydrogenase
-
11beta-hydroxysteroid dehydrogenase
epididymal fat, presence of a functional glucose-6-phosphate-transporter hexose-6-phosphate dehydrogenase 11beta-hydroxysteroid dehydrogenase type 1 system
11beta-hydroxysteroid dehydrogenase
methionine restriction induces isoform 11beta-HSD1 activity in all types of adipose tissue examined, correlating with increased tissue corticosterone. Inverse relationship between 11beta-HSD1 activity and adipocyte size is observed. Methionine restriction additionally increases adipose triglyceride lipase and acetyl-coenzyme A carboxylase protein levels
IMP dehydrogenase
-
isocitrate dehydrogenase (NADP+)
-
phosphogluconate dehydrogenase (NADP+-dependent, decarboxylating)
-
17beta-estradiol 17-dehydrogenase
-
glycerol-3-phosphate dehydrogenase (NAD+)
-
phosphoglycerate dehydrogenase
-
11beta-hydroxysteroid dehydrogenase (NAD+)
-
glutathione peroxidase
pGPx is expressed during bovine adipocyte differentiation, transcriptional control of pGPx in cattle might be carried out by C/EBPdelta
cysteine dioxygenase
-
beta-carotene 15,15'-dioxygenase
-
calcidiol 1-monooxygenase
-
stearoyl-CoA 9-desaturase
-
thyroxine 5-deiodinase
cell culture; isozyme type III
thyroxine 5'-deiodinase
-
thyroxine 5'-deiodinase
isozyme type II
13,14-dehydro-15-oxoprostaglandin 13-reductase
-
acyl-CoA oxidase
-
medium-chain acyl-CoA dehydrogenase
-
all-trans-retinol 13,14-reductase
-
primary-amine oxidase
-
primary-amine oxidase
major SSAO form expressed in mouse adipocytes is encoded by the AOC3 gene; the major SSAO form expressed in mouse adipocytes is encoded by the AOC3 gene
primary-amine oxidase
most of the SSAO found in adipose tissue originates from mature adipocytes
primary-amine oxidase
SSAO inhibition is not sufficient to impair fat deposition. However, combined monoamine oxidase (EC 1.4.3.4) inhibition and SSAO inhibition limits adiposity in non-obese as well as in obese rats
monoamine oxidase
-
monoamine oxidase
in human subcutaneous adipose tissue, the adipocyte-enriched fraction exhibits seven-fold higher amine oxidase activity and contains 3- to 7-fold higher levels of mRNAs encoded by MAO-A and MAO-B genes than the stroma-vascular fraction. MAO-A gene accounts for the majority of the respective MAO and SSAO activities in human adipose tissue. Most of the SSAO and MAO found in adipose tissue originated from mature adipocytes; in human subcutaneous adipose tissue, the adipocyte-enriched fraction exhibits seven-fold higher amine oxidase activity and contains 3- to 7-fold higher levels of mRNAs encoded by MAO-A and MAO-B genes than the stroma-vascular fraction. MAO-A gene accounts for the majority of the respective MAO and SSAO activities in human adipose tissue. Most of the SSAO and MAO found in adipose tissue originated from mature adipocytes
monoamine oxidase
most of the monoamine oxidase found in adipose tissue originates from mature adipocytes
monoamine oxidase
SSAO inhibition is not sufficient to impair fat deposition. Combined MAO and SSAO inhibition limits adiposity in non-obese as well as in obese rats
NAD(P)H oxidase (H2O2-forming)
isoform Nox4 is expressed at high levels in white and brown preadipocytes. Differentiation into adipocytes results in a decrease in their NOX4 mRNA content. In intact adipose tissue, the majority of NOX4 expressing cells are localized within the preadipocyte containing stromal/vascular fracftion. Alterations in NOX4 expression reflects changes in the ratio of adipocyte/interstitial fractions
nicotinamide N-methyltransferase
-
nicotinamide N-methyltransferase
NNMT expression profile, overview
tRNA (cytosine38-C5)-methyltransferase
-
alpha-tubulin N-acetyltransferase
acetylation of alpha-tubulin is up-regulated during adipogenesis, and adipocyte development is dependent on alpha-tubulin acetylation
glycerol-3-phosphate 1-O-acyltransferase
-
glycerol-3-phosphate 1-O-acyltransferase
GPAT3 mRNA is dramatically upregulated during adipocyte differentiation
glycerol-3-phosphate 1-O-acyltransferase
mitochondrial acyltransferase
diacylglycerol O-acyltransferase
isolated from parametrial adipose
carnitine O-palmitoyltransferase
-
1-acylglycerophosphocholine O-acyltransferase
by retrieving LPCAT3 expression pattern from Genevestigator
glycerone-phosphate O-acyltransferase
-
1-acylglycerol-3-phosphate O-acyltransferase
-
1-acylglycerol-3-phosphate O-acyltransferase
isozyme AGPAT2
ATP citrate synthase
-
glycogen(starch) synthase
3T3-L1
nicotinamide phosphoribosyltransferase
-
protein geranylgeranyltransferase type II
-
aspartate transaminase
-
aspartate transaminase
differentiated from 3T3-L1 fibroblasts
hexokinase
very low expression
deoxyguanosine kinase
-
phosphatidylinositol 3-kinase
-
phosphatidylinositol 3-kinase
3T3-L1
phosphatidylinositol 3-kinase
3T3-L1; ,during differentiation of 3T3-L1 cells into adipocytes, isoform p110b is up-regulated approximately 10-fold, expression of p110a is unaltered
phosphatidylinositol 3-kinase
insulin-sensitive mouse 3T3-L1 adipocytes
phosphatidylinositol 3-kinase
primary, obtained from subcutaneous tissue biopsies performed on two lean non-diabetic male subjects
1-phosphatidylinositol-3-phosphate 5-kinase
-
glycerol kinase
There is no difference in glycerol mRNA level between control 3T3-L1 adipocytes and 3T3-L1 Aqp7-RNAi transfected adipocytes, whereas a 4fold enzymatic activation of glycerol kinase is observable in Aqp7 knockout adipocytes (activity assay: 50 mM TrisHCl, pH 7.2, 5 mM ATP, 10 mM MgCl2, 100 mM KCl, 2.5 mM DTT, 4 mM glycerol, 500 microM 3H-glycerol, for 90 min at 37C).
pyruvate kinase
expression of M2-type pyruvate kinase isoenzyme
1-phosphatidylinositol 4-kinase
-
deoxycytidine kinase
-
receptor protein-tyrosine kinase
-
non-specific serine/threonine protein kinase
-
cAMP-dependent protein kinase
-
cGMP-dependent protein kinase
-
protein kinase C
-
beta-adrenergic-receptor kinase
-
[hydroxymethylglutaryl-CoA reductase (NADPH)] kinase
-
[hydroxymethylglutaryl-CoA reductase (NADPH)] kinase
primary
[3-methyl-2-oxobutanoate dehydrogenase (acetyl-transferring)] kinase
-
inositol-hexakisphosphate kinase
-
ethanolamine-phosphate cytidylyltransferase
-
ethanolamine-phosphate cytidylyltransferase
; high Pcyt2 mRNA content. Negligible amount of Pcyt2beta mRNA, high amount of Pcyt2alpha mRNA
ethanolaminephosphotransferase
-
diacylglycerol cholinephosphotransferase
-
thiosulfate sulfurtransferase
-
estrone sulfotransferase
-
acylglycerol lipase
-
triacylglycerol lipase
hormone-sensitive lipase is negatively regulated by perilipin, lipolysis of storage triacylglycerides
triacylglycerol lipase
hormone-sensitive lipase, lipolysis of storage triacylglycerides
1-alkyl-2-acetylglycerophosphocholine esterase
-
hormone-sensitive lipase
adipocytes retrovirally engineered from murine embryonic fibroblasts
hormone-sensitive lipase
dydrogesterone and norethisterone regulate expression of hormone-sensitive lipase in human subcutaneous abdominal adipocytes
hormone-sensitive lipase
women with polycystic ovary syndrome or obesity have significantly decreased expression of hormone-sensitive lipase. Knock down of hormone-sensitive lipase by RNAi reduces basal and catecholamine-induced lipolysis. Treatment of mesenchymal stem cells with a selective inhibitor of hormone-sensitive lipase during and/or after differentiation in adipocytes reduces basal lipolysis by 50%
protein-serine/threonine phosphatase
-
phosphoinositide 5-phosphatase
-
phosphoinositide 5-phosphatase
72-5ptase is expressed in differentiated 3T3-L1 adipocytes
fructose-2,6-bisphosphate 2-phosphatase
inducible expression of isozyme PFKFB3
fructose-2,6-bisphosphate 2-phosphatase
inducible expression of isozyme PFKFB3, visceral fat cell
protein-tyrosine-phosphatase
-
phosphatidylinositol-3,4,5-trisphosphate 3-phosphatase
-
3',5'-cyclic-nucleotide phosphodiesterase
-
3',5'-cyclic-nucleotide phosphodiesterase
primary
alkylglycerophosphoethanolamine phosphodiesterase
-
Results 1 - 100 of 135 > >>