1.3.5.5: 15-cis-phytoene desaturase

This is an abbreviated version!
For detailed information about 15-cis-phytoene desaturase, go to the full flat file.

Reaction

15,9'-dicis-phytofluene
+
plastoquinone
=
9,15,9'-tricis-zeta-carotene
+
plastoquinol

Synonyms

BoPDS, CitPDS1, CrtP, PDS, PDS1, PDS3

ECTree

     1 Oxidoreductases
         1.3 Acting on the CH-CH group of donors
             1.3.5 With a quinone or related compound as acceptor
                1.3.5.5 15-cis-phytoene desaturase

Expression

Expression on EC 1.3.5.5 - 15-cis-phytoene desaturase

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EXPRESSION
ORGANISM
UNIPROT
LITERATURE
although carotenoid accumulation is strongly induced during flower development, only very low concentrations of phytoene desaturase transcripts are detectable, while the corresponding protein accumulates in low, but measurable amounts, appearing in soluble and membrane-bound states
-
Pds is constitutively expressed. Endosperm carotenogenesis is not regulated by increasing the level of phytoene desaturase transcripts
presence of 5-chloro-3-methyl-4-nitro-1H-pyrazole activates phytoene desaturase (Pds) and zeta-carotene desaturase Zds gene expression from early to midmaturation stages of fruit
regulation of phytoene desaturase occurs primarily at the mRNA level, most likely by transcriptional control. The enzyme is induced in response to stress conditions leading to biosynthesis of secondary carotenoids
-
the CitPDS1 transcript in the juice sacs/segment epidermis (edible part) is at a low level in the young fruit, and it increases toward maturation. In the peel the level of the CitPDS1 transcript remains constant after an increase in July
there is no significant induction of phytoene desaturase gene expression specific to white tissue, indicating that phytoene desaturase expression is independent of the pigment status of the cells
-
upon transfer to high light, the transcript levels of all investigated carotenogenic genes including those coding for phytoene synthase, phytoene desaturase and both ketolases are increased. Transcription changes proceed via binding of the transcription factor NtcA to the promoter regions of the carotenogenic genes. The binding is absolutely dependent on the presence of reductants and oxo-glutarate and inhibited by 3-(3, 4-dichlorophenyl)-1,1-dimethyl urea