EC Number |
Inhibitors |
Structure |
---|
1.13.11.69 | more |
no evidence for feedback regulation |
|
1.13.11.69 | more |
AtCCD8 is inhibited in a time-dependent fashion by hydroxamic acids N-[(4-fluorophenyl)methyl]-N-hydroxy-2-(4-hydroxyphenyl)acetamide, N-[(4-fluorophenyl)methyl]-N-hydroxy-2-(4-methoxyphenyl)acetamide, N-benzyl-2-(3,4-dimethoxyphenyl)-N-hydroxyacetamide and 2-(3,4-dimethoxyphenyl)-N-[(4-fluorophenyl)methyl]-N-hydroxyacetamide with over 95% inhibition at 0.10 mM, hydroxamic acids acids N-[(4-fluorophenyl)methyl]-N-hydroxy-2-(4-hydroxyphenyl)acetamide, N-[(4-fluorophenyl)methyl]-N-hydroxy-2-(4-methoxyphenyl)acetamide, N-benzyl-2-(3,4-dimethoxyphenyl)-N-hydroxyacetamide and 2-(3,4-dimethoxyphenyl)-N-[(4-fluorophenyl)methyl]-N-hydroxyacetamide cause a shoot branching phenotype in Arabidopsis thaliana. Selective inhibition of CCD8 is observed using hydroxamic acids N-hydroxy-3-(4-methoxyphenyl)propanamide and N-[(4-fluorophenyl)methyl]-N-hydroxy-3-(4-methoxyphenyl)propanamide. No inhibition by N1-[(4-fluorophenyl)methyl]-N1-hydroxy-N4-[(4-methoxyphenyl)methyl]butanediamide |
|
1.13.11.69 | cytokinin |
- |
|
1.13.11.69 | abamine |
over 95% inhibition at 0.1 mM |
|
1.13.11.69 | 2-(2H-1,3-benzodioxol-5-yl)-N-[(4-fluorophenyl)methyl]-N-hydroxyacetamide |
over 95% inhibition at 0.1 mM |
|
1.13.11.69 | (2E)-3-(3,4-dimethoxyphenyl)-N-hydroxyprop-2-enamide |
over 95% inhibition at 0.1 mM |
|
1.13.11.69 | 3-(3,4-dimethoxyphenyl)-N-hydroxypropanamide |
78% inhibition at 0.1 mM |
|
1.13.11.69 | (2E)-N-hydroxy-3-(4-methoxyphenyl)prop-2-enamide |
over 95% inhibition at 0.1 mM |
|
1.13.11.69 | N-hydroxy-3-(4-methoxyphenyl)propanamide |
over 95% inhibition at 0.1 mM |
|
1.13.11.69 | 3-(3,4-dimethoxyphenyl)-N-hydroxy-N-octylpropanamide |
over 95% inhibition at 0.1 mM |
|