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Results 1 - 8 of 8
EC Number Disease PubMed ID Title of Publication Category Confidence Level
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Infections 25525797 Rhinovirus uses a phosphatidylinositol 4-phosphate/cholesterol counter-current for the formation of replication compartments at the ER-Golgi interface. unassigned 0
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Myopathies, Structural, Congenital 22068590 Modeling the human MTM1 p.R69C mutation in murine Mtm1 results in exon 4 skipping and a less severe myotubular myopathy phenotype. causal interaction
unassigned
3
0
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Neoplasms 24091101 SHIP2 signaling in normal and pathological situations: Its impact on cell proliferation. causal interaction
ongoing research
unassigned
3
4
0
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Neoplasms 27405757 Controlling PTEN (Phosphatase and Tensin Homolog) Stability: A DOMINANT ROLE FOR LYSINE 66. causal interaction
diagnostic usage
unassigned
3
3
0
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Neuromuscular Diseases 22068590 Modeling the human MTM1 p.R69C mutation in murine Mtm1 results in exon 4 skipping and a less severe myotubular myopathy phenotype. causal interaction
unassigned
3
0
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Osteosarcoma 7708721 A domain of human immunodeficiency virus type 1 Vpr containing repeated H(S/F)RIG amino acid motifs causes cell growth arrest and structural defects. unassigned 0
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Rhabdomyosarcoma 7708721 A domain of human immunodeficiency virus type 1 Vpr containing repeated H(S/F)RIG amino acid motifs causes cell growth arrest and structural defects. unassigned 0
Display the word mapDisplay the reaction diagram Show all sequences 3.1.3.B4Starvation 17908202 Growth control of Golgi phosphoinositides by reciprocal localization of sac1 lipid phosphatase and pik1 4-kinase. unassigned 0
Results 1 - 8 of 8