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EC Number
Disease
PubMed ID
Title of Publication
Category
Confidence Level
Acrocephalosyndactylia
Twist haploinsufficiency in Saethre-Chotzen syndrome induces calvarial osteoblast apoptosis due to increased TNFalpha expression and caspase-2 activation.
causal interaction
1
Acrocephalosyndactylia
Twist haploinsufficiency in Saethre-Chotzen syndrome induces calvarial osteoblast apoptosis due to increased TNFalpha expression and caspase-2 activation.
therapeutic application
1
Acrocephalosyndactylia
Twist haploinsufficiency in Saethre-Chotzen syndrome induces calvarial osteoblast apoptosis due to increased TNFalpha expression and caspase-2 activation.
unassigned
0
Adenocarcinoma
Attenuation of the ELAV1-like protein HuR sensitizes adenocarcinoma cells to the intrinsic apoptotic pathway by increasing the translation of caspase-2L.
therapeutic application
1
Adenocarcinoma
Attenuation of the ELAV1-like protein HuR sensitizes adenocarcinoma cells to the intrinsic apoptotic pathway by increasing the translation of caspase-2L.
ongoing research
3
Adenocarcinoma
Attenuation of the ELAV1-like protein HuR sensitizes adenocarcinoma cells to the intrinsic apoptotic pathway by increasing the translation of caspase-2L.
unassigned
0
Adenocarcinoma of Lung
c-Myc and caspase-2 are involved in activating Bax during cytotoxic drug-induced apoptosis.
diagnostic usage
2
Adenocarcinoma of Lung
Emodin induces apoptosis in human lung adenocarcinoma cells through a reactive oxygen species-dependent mitochondrial signaling pathway.
therapeutic application
2
Adenocarcinoma of Lung
Emodin induces apoptosis in human lung adenocarcinoma cells through a reactive oxygen species-dependent mitochondrial signaling pathway.
ongoing research
4
Adenocarcinoma of Lung
c-Myc and caspase-2 are involved in activating Bax during cytotoxic drug-induced apoptosis.
ongoing research
4
Results 1 - 10 of 998 > >>