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Results 1 - 10 of 269 > >>
EC Number
Disease
PubMed ID
Title of Publication
Category
Confidence Level
Blast Crisis
c-MYB is a transcriptional regulator of ESPL1/Separase in BCR-ABL-positive chronic myeloid leukemia.
causal interaction
1
Blast Crisis
Clonal Evolution and Blast Crisis Correlate with Enhanced Proteolytic Activity of Separase in BCR-ABL b3a2 Fusion Type CML under Imatinib Therapy.
therapeutic application
3
Blast Crisis
c-MYB is a transcriptional regulator of ESPL1/Separase in BCR-ABL-positive chronic myeloid leukemia.
therapeutic application
1
Blast Crisis
Clonal Evolution and Blast Crisis Correlate with Enhanced Proteolytic Activity of Separase in BCR-ABL b3a2 Fusion Type CML under Imatinib Therapy.
unassigned
0
Blast Crisis
c-MYB is a transcriptional regulator of ESPL1/Separase in BCR-ABL-positive chronic myeloid leukemia.
unassigned
0
Breast Neoplasms
Separase is a marker for prognosis and mitotic activity in breast cancer.
causal interaction
3
Breast Neoplasms
Overexpression of Separase induces aneuploidy and mammary tumorigenesis.
causal interaction
4
Breast Neoplasms
MMTV-Espl1 transgenic mice develop aneuploid, estrogen receptor alpha (ER?)-positive mammary adenocarcinomas.
causal interaction
3
Breast Neoplasms
Biology and insights into the role of cohesin protease separase in human malignancies.
causal interaction
3
Breast Neoplasms
Separase is a marker for prognosis and mitotic activity in breast cancer.
diagnostic usage
4
Results 1 - 10 of 269 > >>