EC Number   |
General Information |
Reference |
|---|
    6.6.1.1 | malfunction |
a rtl1 mutant allele of the CHLH gene in Arabidopsis thaliana shows ABA-insensitive phenotypes in both stomatal movement and seed germination |
728494 |
| 6.6.1.1 | malfunction |
CHLH RNAi-silenced plants show phenotypes in which stomatal movements are insensitive to abscisic acid |
716044 |
| 6.6.1.1 | malfunction |
mutant alleles of GUN4 and Mg-chelatase genes cause sensitivity to intense light |
716520 |
| 6.6.1.1 | malfunction |
overexpression of CHLD gene shows wild-type ABA sensitivity in Arabidopsis |
728494 |
| 6.6.1.1 | metabolism |
magnesium chelatase catalyses the first committed step of chlorophyll biosynthesis |
715033 |
| 6.6.1.1 | metabolism |
Mg-chelatase catalyzes the first step of the chlorophyll biosynthetic pathway |
710579 |
| 6.6.1.1 | physiological function |
Mg-chelatase H subunit affects abscisic acid signaling in stomatal guard cells, but is not an abscisic acid receptor |
716044 |
| 6.6.1.1 | physiological function |
the enzyme has a key role in chlorophyll biosynthesis |
748471 |
| 6.6.1.1 | physiological function |
the magnesium-protoporphyrin IX chelatase H subunit (CHLH/ABAR) functions as a receptor for abscisic acid in Arabidopsis thaliana. ABAR is a key component connecting the circadian clock with abscisic acid-mediated plant responses to drought. ABAR antagonizes negative abscisic acid signaling regulators WRKYs to derepress abscisic acid-responsive genes |
716515 |
| 6.6.1.1 | physiological function |
the Mg-chelatase complex and Mg-protoporphyrin IX methyltransferase specifically affect abscisic acid signaling in the control of stomatal aperture |
748487 |
