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Commentary
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3.4.11.20
malfunction
inhibition caused swelling of the parasite digestive vacuole and prevented proteolysis of hemoglobin derived oligopeptides, likely starving the parasite resulting in death
718330
3.4.11.20
malfunction
inhibition is lethal to parasites early in the life cycle, prior to the onset of hemoglobin degradation
718330
3.4.11.20
physiological function
synthesized as the precursor p120 form, targeted to the parasitophorous vacuole via the parasite endoplasmic reticulum/Golgi, where it is converted into the transient p96 form; p96 form is eventually redirected into the parasite to be converted into the processed p68 form that is only marginally delivered to the parasite foodvacuole
718071
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