EC Number   |
General Information |
Reference |
|---|
   2.1.1.222 | malfunction |
after 1 h of H2O2 treatment, the viability of the strain defective in ubiG is reduced 28fold compared to the corresponding wild-type. When treated with CuSO4, the viability of the the strain defective in ubiG (HW271), is reduced 4.4fold after 30 min compared to its isogenic wild-type |
705549 |
| 2.1.1.222 | metabolism |
the enzyme catalyzes both of the two O-methyltransfer steps in CoQ10 biosynthesis |
733281 |
| 2.1.1.222 | metabolism |
the enzyme is one of the key enzymes in the ubiquinone modification pathway |
734623 |
| 2.1.1.222 | metabolism |
the enzyme is required for both O-methylation steps in ubiquinone biosynthesis |
718512 |
| 2.1.1.222 | physiological function |
the membrane-binding region of UbiG gates the entrance of methyl donor. When bound with liposome, UbiG displays an enhanced binding ability toward the methyl donor product S-adenosylhomocysteine |
758360 |
| 2.1.1.222 | physiological function |
the ubiG gene product is essential for growth on glycerol minimal medium |
704270 |
