EC Number |
General Information |
Reference |
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1.14.99.22 | malfunction |
enzyme knockdown causes larval lethality, delayed development and affects pupation |
745079 |
1.14.99.22 | malfunction |
RNAi-mediated knockdown of Shade negatively affects ecdysone-20-hydroxylation in the desert locust |
728083 |
1.14.99.22 | more |
in the pupa of the silkworm, Bombyx mori, the rectal sac is an essential organ that operates as a repository for degraded ecdysteroids, and it can be distended by administration of 20-hydroxyecdysone |
715820 |
1.14.99.22 | more |
physiological and developmental significance of the mitochondrial, NADPH-forming energy-linked transhydrogenations, that catalyzes a reversible hydride ion transfer between NADP(H) and NAD(H), for E20-M activity. NADPH-forming activity occurs as a nonenergy- or energy-linked activity with energy for the latter derived from either electron transport-dependent NADH or succinate utilization, or ATP hydrolysis by Mg2+-dependent ATPase, overview |
715821 |
1.14.99.22 | physiological function |
essential need for conversion of the molting hormone, ecdysone, to its physiologically active form, 20-hydroxyecdysone by E20-M during the post-embryonic development |
715821 |
1.14.99.22 | physiological function |
in the desert locust, the Halloween gene SgShd plays a role in the biosynthesis of 20-hydroxyecdysone |
728083 |