EC Number |
General Information |
Reference |
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1.13.11.4 | malfunction |
disruption of dsmD results in a significant decline in the degradation rates of 3-chlorogentisate and the herbicide dicamba but has no effect on the degradation of gentisate. The disruption of both dsmD and gtdA leads to the inability to degrade 3-chlorogentisate and gentisate |
-, 764960 |
1.13.11.4 | metabolism |
narI is involved in salicylate, i.e. 3-hydroxybenzoate, and gentisate catabolism, organization of the 3HBA-gentisate catabolic cluster, overview |
-, 710986 |
1.13.11.4 | metabolism |
strain CJ2 metabolizes naphthalene via the gentisate pathway using gentisate 1,2-dioxygenase, patterns in regulation, transcription and enzyme activity, overview. The nagR regulator controls the third catabolic gene cluster |
725905 |
1.13.11.4 | metabolism |
the gene genH belongs to the group involved in naphthalene and salicylate degradation, by the nar and gen gene clusters, respectively, overview |
-, 710974 |
1.13.11.4 | more |
binding modes for salicylate and gentisate |
-, 724975 |
1.13.11.4 | more |
strain NCIMB 12038 utilizes naphthalene as a sole source of carbon and energy, and degrades naphthalene via salicylate and gentisate. The strain can also use salicylate, i.e. 3-hydroxybenzoate, as sole source of energy and carbon |
-, 710986 |
1.13.11.4 | physiological function |
although all three nagI genes in strain CJ2 influence naphthalene degradation, the nagI3 gene plays the most significant role. NagI3 may prevent toxic intracellular levels of gentisate from accumulating in CJ2 cells |
725905 |
1.13.11.4 | physiological function |
DsmD is specifically responsible for cleavage of 3-chlorogentisate, a key intermediate in the catabolism of the herbicide dicamba in Rhizorhabdus dicambivorans Ndbn-20 |
-, 764960 |
1.13.11.4 | physiological function |
the enzyme is involved in the 3-phenylpropionic acid degradation pathway |
727451 |