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Results 1 - 5 of 5
EC Number
General Information
Commentary
Reference
malfunction
RNAi-mediated depletion of the enzyme effectively abrogates ectopic E2F1-induced apoptosis, coupled with reduced lysosomal membrane permeabilization and mitochondrial membrane depolarization. Enzyme knockdown also inhibits apoptosis mediated by the endogenous E2F1 activated by DNA damage. Enzyme depletion in cancer cells results in inhibition of histone deacetylase inhibitor-induced apoptosis
metabolism
cathepsin V participates in the production of enkephalin and neuropeptide Y neurotransmitters in secretory vesicles
physiological function
cathepsin like 2 is required for E2F1-induced apoptosis by engaging lysosomal membrane permeabilization. Its level also modulates the cellular sensitivity to histone deacetylase inhibitor
physiological function
CTSV is involved in the breakdown of corneodesmosomal proteins
physiological function
expression of CTSV rescues the reduced frequency of CD4+ T cells in cysteine protease cathepsin L-deficient mice with H-2b haplotype
Results 1 - 5 of 5