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General Information
an enzyme deletion mutant exhibits the ability to utilize glucose for succinic acid production efficiently
physiological function
complementation of an ach1 mutant with the coaT gene reverses the growth defect on acetate confirming the in vivo function of Ach1p as a CoA-transferase. Growth of an ach1p deletion mutant on the carboxylic acids lactate and pyruvate reveals no phenotype
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