3.4.24.82	Abortion, Spontaneous	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31010360&form=6&db=m	Decreased ADAMTS-1, -9 and -20 levels in women with endometrial polyps: a possible link between extracellular matrix proteases and endometrial pathologies	diagnostic usage,unassigned	4,0	
3.4.24.82	Abscess	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31063202&form=6&db=m	Histopathologic and immunohistochemical investigations of abscess fluid cells formed in the palato-gingival area.	causal interaction,ongoing research,unassigned	3,4,0	
3.4.24.82	Acute Coronary Syndrome	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19944557&form=6&db=m	ADAMTS4 level in patients with stable coronary artery disease and acute coronary syndromes.	causal interaction,diagnostic usage,ongoing research,therapeutic application	4,4,4,1	
3.4.24.82	Acute Coronary Syndrome	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20625753&form=6&db=m	Elevated level of ADAMTS4 in plasma and peripheral monocytes from patients with acute coronary syndrome.	causal interaction,diagnostic usage,ongoing research,unassigned	4,4,4,0	
3.4.24.82	Acute Coronary Syndrome	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27491335&form=6&db=m	Loss of ADAMTS4 reduces high fat diet-induced atherosclerosis and enhances plaque stability in ApoE(-/-) mice.	causal interaction,diagnostic usage,ongoing research,therapeutic application	4,4,4,4	
3.4.24.82	adamts-4 endopeptidase deficiency	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28955046&form=6&db=m	Critical Role of ADAMTS-4 in the Development of Sporadic Aortic Aneurysm and Dissection in Mice.	causal interaction,ongoing research,unassigned	2,4,0	
3.4.24.82	Alzheimer Disease	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28084617&form=6&db=m	ADAMTS-4 in central nervous system pathologies.	causal interaction,ongoing research,therapeutic application,unassigned	3,2,1,0	
3.4.24.82	Alzheimer Disease	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30426203&form=6&db=m	The metalloprotease ADAMTS4 generates N-truncated A?4-x species and marks oligodendrocytes as a source of amyloidogenic peptides in Alzheimer's disease.	causal interaction,therapeutic application,unassigned	1,1,0	
3.4.24.82	Amyotrophic Lateral Sclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26809777&form=6&db=m	ADAMTS-4 promotes neurodegeneration in a mouse model of amyotrophic lateral sclerosis.	ongoing research,unassigned	4,0	
3.4.24.82	Amyotrophic Lateral Sclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26858022&form=6&db=m	Erratum to: ADAMTS-4 promotes neurodegeneration in a mouse model of amyotrophic lateral sclerosis.	ongoing research,unassigned	2,0