3.2.1.84 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28929344&form=6&db=m Glucosidase II beta subunit (GluII?) plays a role in autophagy and apoptosis regulation in lung carcinoma cells in a p53-dependent manner. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,1 3.2.1.84 Carcinoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28929344&form=6&db=m Glucosidase II beta subunit (GluII?) plays a role in autophagy and apoptosis regulation in lung carcinoma cells in a p53-dependent manner. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,3,3,1 3.2.1.84 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3546312&form=6&db=m Glucosidase II, a protein of the endoplasmic reticulum with high mannose oligosaccharide chains and a rapid turnover. ongoing research,unassigned 3,0 3.2.1.84 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17905686&form=6&db=m [Effect of green tea flavanols on the functions of the endoplasmic reticulum.] causal interaction,unassigned 3,0 3.2.1.84 Carcinoma, Hepatocellular http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18765294&form=6&db=m Endoplasmic reticulum stress underlying the pro-apoptotic effect of epigallocatechin gallate in mouse hepatoma cells. ongoing research,unassigned 4,0 3.2.1.84 Dental Plaque http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23520578&form=6&db=m Purification and properties of an ?-(1 ? 3)-glucanase (EC 3.2.1.84) from Trichoderma harzianum and its use for reduction of artificial dental plaque accumulation. unassigned - 3.2.1.84 Diabetes Mellitus, Type 2 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32232844&form=6&db=m Metabolic syndrome perturbs deglucosylation and reglucosylation in the glycoprotein folding cycle. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,4,0 3.2.1.84 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=2472115&form=6&db=m Antibody-independent, complement-mediated enhancement of HIV-1 infection by mannosidase I and II inhibitors. causal interaction,therapeutic application,unassigned 1,4,0 3.2.1.84 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27842496&form=6&db=m Proteomics analysis reveals novel host molecular mechanisms associated with thermotherapy of 'Ca. Liberibacter asiaticus'-infected citrus plants. therapeutic application,unassigned 1,0 3.2.1.84 Liver Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15133510&form=6&db=m Mutations in SEC63 cause autosomal dominant polycystic liver disease. unassigned - 3.2.1.84 Liver Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15188177&form=6&db=m Molecular characterization of hepatocystin, the protein that is defective in autosomal dominant polycystic liver disease. causal interaction,diagnostic usage,unassigned 3,1,0 3.2.1.84 Liver Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15649821&form=6&db=m Polycystic liver disease is a disorder of cotranslational protein processing. unassigned - 3.2.1.84 Liver Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24719335&form=6&db=m N-glycosylation determines the abundance of the transient receptor potential channel TRPP2. unassigned - 3.2.1.84 Lung Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31316108&form=6&db=m Knockout of glucosidase II beta subunit inhibits growth and metastatic potential of lung cancer cells by inhibiting receptor tyrosine kinase activities. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,1 3.2.1.84 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1328242&form=6&db=m Identification of a novel mechanism for the removal of glucose residues from high mannose-type oligosaccharides. ongoing research,unassigned 3,0 3.2.1.84 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3881423&form=6&db=m Identity of neutral alpha-glucosidase AB and the glycoprotein processing enzyme glucosidase II. Biochemical and genetic studies. causal interaction,unassigned 3,0 3.2.1.84 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=3924028&form=6&db=m The effect of castanospermine on the oligosaccharide structures of glycoproteins from lymphoma cell lines. ongoing research,unassigned 4,0 3.2.1.84 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=7050108&form=6&db=m A lectin-resistant mouse lymphoma cell line is deficient in glucosidase II, a glycoprotein-processing enzyme. unassigned - 3.2.1.84 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9254044&form=6&db=m Expression of a cDNA encoding the glucose trimming enzyme glucosidase II in CHO cells and molecular characterization of the enzyme deficiency in a mutant mouse lymphoma cell line. ongoing research,unassigned 4,0 3.2.1.84 Lymphoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=11159920&form=6&db=m N-linked oligosaccharide processing enzyme glucosidase II produces 1,5-anhydrofructose as a side product. ongoing research,unassigned 3,0 3.2.1.84 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32232844&form=6&db=m Metabolic syndrome perturbs deglucosylation and reglucosylation in the glycoprotein folding cycle. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,4,0 3.2.1.84 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22028469&form=6&db=m Proteomic and functional genomic landscape of receptor tyrosine kinase and ras to extracellular signal-regulated kinase signaling. unassigned - 3.2.1.84 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24008518&form=6&db=m Glucosidase II exhibits similarity to the p53 tumor suppressor in regards to structure and behavior in response to stress signals: A potential novel cancer biomarker. causal interaction,therapeutic application,unassigned 4,1,0 3.2.1.84 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31316108&form=6&db=m Knockout of glucosidase II beta subunit inhibits growth and metastatic potential of lung cancer cells by inhibiting receptor tyrosine kinase activities. causal interaction,diagnostic usage,ongoing research,therapeutic application 3,3,3,1 3.2.1.84 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32232844&form=6&db=m Metabolic syndrome perturbs deglucosylation and reglucosylation in the glycoprotein folding cycle. causal interaction,diagnostic usage,ongoing research,unassigned 3,2,4,0 3.2.1.84 Polycystic Kidney Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30792735&form=6&db=m GANAB and PKD1 Variations in a 12 Years Old Female Patient With Early Onset of Autosomal Dominant Polycystic Kidney Disease. causal interaction,unassigned 2,0 3.2.1.84 Polycystic Kidney, Autosomal Dominant http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30792735&form=6&db=m GANAB and PKD1 Variations in a 12 Years Old Female Patient With Early Onset of Autosomal Dominant Polycystic Kidney Disease. causal interaction,unassigned 2,0 3.2.1.84 Vesicular Stomatitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=1328242&form=6&db=m Identification of a novel mechanism for the removal of glucose residues from high mannose-type oligosaccharides. ongoing research,unassigned 3,0