1.11.1.11	H2O2	-	
1.11.1.11	methyl jasmonate	knockout mutant plants lacking Apx1 show high sensitivity to wounding and methyl jasmonate treatment. In the leaves of wild-type plants, H2O2 accumulates only in the vicinity of the wound, while in the leaves of the knockout mutant plants it accumulates extensively from damaged to undamaged regions. During methyl jasmonate treatment, the levels of H2O2 are much higher in the leaves of Apx1 knockout plants	
1.11.1.11	additional information	S-nitrosylation and S-sulfhydration positively regulate the peroxidase activity, whereas no effects are observed on the chaperone function and the oligomeric status of AtAPX1	
1.11.1.11	nitric oxide	causes an increase in the total enzymatic activity of ascorbate peroxidase. The nitric oxide-induced changes in ascorbate peroxidase enzymatic activity are coupled to altered nodule H2O2 content. Enzymatic activity of the largest isoform, Apx1 is upregulated by 11% in response to 5 microM of nitric oxide donor DETA/NO and 21% in response to 10 microM DETA/NO. 5 microM of DETA/NO increase the enzymatic activity of the second largest isoform Apx2 by about 57%. Treatment of nodulated soybean with 5 microM DETA/NO upregulates the smallest nodule isoform Apx3 enzymatic activity by 228% compared to untreated controls	
1.11.1.11	phenanthrene	exposure to 0.5 microM phenanthrene results in significant increase in the levels of both enzymatic and non-enzymatic antioxidants, with the levels of total glutathione and ascorbate doubling, the activitiy of APOX increasing by 2fold after 72 h of exposure to phenanthrene	
1.11.1.11	Zinc	-