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Literature summary extracted from

  • Sasaki, Y.; Araki, T.; Milbrandt, J.
    Stimulation of nicotinamide adenine dinucleotide biosynthetic pathways delays axonal degeneration after axotomy (2006), J. Neurosci., 26, 8484-8491.
    View publication on PubMedView publication on EuropePMC

Application

EC Number Application Comment Organism
2.7.7.1 additional information The expression of Nmnat1 itself is sufficient to protect axons in an in vitro Wallerian degeneration assay, suggesting that increased NAD production is crucial for axonal protection. Mus musculus

Localization

EC Number Localization Comment Organism GeneOntology No. Textmining
2.7.7.1 cytoplasm Nmnat1 Mus musculus 5737
-
2.7.7.1 mitochondrion NMNAT3 Mus musculus 5739
-
2.7.7.1 nucleus Nmnat1 Mus musculus 5634
-

Natural Substrates/ Products (Substrates)

EC Number Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
2.7.7.1 ATP + nicotinamide ribonucleotide Mus musculus
-
diphosphate + NAD+
-
?
2.7.7.1 nicotinic acid mononucleotide + ATP Mus musculus
-
nicotinic acid adenine dinucleotide + diphosphate
-
?

Organism

EC Number Organism UniProt Comment Textmining
2.7.7.1 Mus musculus Q99JR6
-
-
2.7.7.1 Mus musculus Q9EPA7
-
-

Substrates and Products (Substrate)

EC Number Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
2.7.7.1 ATP + nicotinamide ribonucleotide
-
Mus musculus diphosphate + NAD+
-
?
2.7.7.1 nicotinic acid mononucleotide + ATP
-
Mus musculus nicotinic acid adenine dinucleotide + diphosphate
-
?

Synonyms

EC Number Synonyms Comment Organism
2.7.7.1 nicotinamide mononucleotide adenylyltransferase 1
-
Mus musculus
2.7.7.1 NMNAT1
-
Mus musculus
2.7.7.1 NMNAT3
-
Mus musculus