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Literature summary for 3.5.4.37 extracted from
- Specificity of ADAR-mediated RNA editing in newly identified targets (2008), RNA, 14, 1110-1118.
Natural Substrates/ Products (Substrates)
| Natural Substrates | Organism | Comment (Nat. Sub.) | Natural Products | Comment (Nat. Pro.) | Rev. | Reac. |
|---|---|---|---|---|---|---|
| adenine in double-stranded RNA + H2O | Mus musculus | editing of Blcap, FlnA, and some sites within B1 and B2 SINEs clearly depends on ADAR1 | hypoxanthine in double-stranded RNA + NH3 | - |
? |  |
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Mus musculus | - |
- |
- |
Substrates and Products (Substrate)
| Substrates | Comment Substrates | Organism | Products | Comment (Products) | Rev. | Reac. |
|---|---|---|---|---|---|---|
| adenine in double-stranded RNA + H2O | editing of Blcap, FlnA, and some sites within B1 and B2 SINEs clearly depends on ADAR1 | Mus musculus | hypoxanthine in double-stranded RNA + NH3 | - |
? | |
Synonyms
| Synonyms | Comment | Organism |
|---|---|---|
| ADAR1 | - |
Mus musculus |
| ADAR2 | - |
Mus musculus |
General Information
| General Information | Comment | Organism |
|---|---|---|
| malfunction | the ADAR2 knockout phenotype can be attributed to the lack of editing of the GluR-B receptor. ADAR1 deficiency results in an embryonic lethal phenotype | Mus musculus |
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