Any feedback?
Please rate this page
(literature.php)
(0/150)

BRENDA support

Literature summary for 3.4.24.71 extracted from

  • Whyteside, A.R.; Hinsley, E.E.; Lambert, L.A.; McDermott, P.J.; Turner, A.J.
    ECE-1 influences prostate cancer cell invasion via ET-1-mediated FAK phosphorylation and ET-1-independent mechanisms (2010), Can. J. Physiol. Pharmacol., 88, 850-854.
    View publication on PubMed

Inhibitors

Inhibitors Comment Organism Structure
ECEi addition of an ECE-1 specific inhibitor (ECEi) to PC-3 cells reduces phosphorylation of focal adhesion kinase Homo sapiens

Organism

Organism UniProt Comment Textmining
Homo sapiens
-
-
-

Source Tissue

Source Tissue Comment Organism Textmining
PC-3 cell
-
Homo sapiens
-
PNT-1A cell
-
Homo sapiens
-

Synonyms

Synonyms Comment Organism
ECE-1
-
Homo sapiens

Expression

Organism Comment Expression
Homo sapiens siRNA-mediated knockdown of ECE-1 results in a significant reduction in FAK phosphorylation down
Homo sapiens transient ECE-1 overexpression in PNT1-a cells increases FAK phosphorylation up

General Information

General Information Comment Organism
malfunction siRNA-mediated knockdown of ECE-1 results in a significant reduction in FAK phosphorylation. Transient ECE-1 overexpression in PNT1-a cells increases FAK phosphorylation Homo sapiens
physiological function ECE-1 influences PC cell invasion via both ET-1-mediated FAK phosphorylation and ET-1 independent mechanisms Homo sapiens