Literature summary for 3.4.24.7 extracted from

Lin, X.; Jo, H.; Ishii, T.M.; Fujita, M.; Fu, M.; Tambara, K.; Yamamoto, M.; Tabata, Y.; Komeda, M.; Matsuoka, S.
Controlled release of matrix metalloproteinase-1 plasmid DNA prevents left ventricular remodeling in chronic myocardial infarction of rats (2009), Circ. J., 73, 2315-2321.

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Protein Variants

Protein Variants	Comment	Organism
additional information	transfection of rat myocard with a plasmid encoding MMP-1, DNA release and MMP1 expression, effects on myocard remodeling, overview. MMP-1 expression increases myocyte shortening and reduces Na+-Ca2+ exchange current, it decreases myocardial fibrosis and improves cardiac remodeling and function	Rattus norvegicus

Inhibitors

Inhibitors	Comment	Organism	Structure
TIMP	-	Rattus norvegicus

Organism

Organism	UniProt	Comment	Textmining
Rattus norvegicus	-	male Lewis rats	-

Source Tissue

Source Tissue	Comment	Organism	Textmining
left ventricle	-	Rattus norvegicus	-
myocardium	-	Rattus norvegicus	-

Synonyms

Synonyms	Comment	Organism
matrix metalloproteinase-1	-	Rattus norvegicus
MMP-1	-	Rattus norvegicus

General Information

General Information	Comment	Organism
physiological function	transfection of a plasmid expression of MMP-1 into myocard after infarction increases myocyte shortening and reduces Na+-Ca2+ exchange current, it decreases myocardial fibrosis and improves cardiac remodeling and function	Rattus norvegicus

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Release 2023.1 (January 2023)