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Literature summary for 3.4.14.5 extracted from

  • Preller, V.; Gerber, A.; Wrenger, S.; Togni, M.; Marguet, D.; Tadje, J.; Lendeckel, U.; Roecken, C.; Faust, J.; Neubert, K.; Schraven, B.; Martin, R.; Ansorge, S.; Brocke, S.; Reinhold, D.
    TGF-beta1-mediated control of central nervous system inflammation and autoimmunity through the inhibitory receptor CD26 (2007), J. Immunol., 178, 4632-4640.
    View publication on PubMed

Application

Application Comment Organism
medicine CD26/DP IV is a nonredundant inhibitory receptor controlling T cell activation and Th1-mediated autoimmunity, and may have important therapeutic implications for the treatment of autoimmune CNS disease Mus musculus

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
additional information Mus musculus CD26/DP IV is a nonredundant inhibitory receptor controlling T cell activation and Th1-mediated autoimmunity ?
-
?

Organism

Organism UniProt Comment Textmining
Mus musculus
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-
-

Source Tissue

Source Tissue Comment Organism Textmining

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
additional information CD26/DP IV is a nonredundant inhibitory receptor controlling T cell activation and Th1-mediated autoimmunity Mus musculus ?
-
?

Synonyms

Synonyms Comment Organism
CD26/dipeptidyl peptidase
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Mus musculus
CD26/DP IV
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Mus musculus