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Literature summary for 3.1.1.77 extracted from

  • Yang, W.; Yaggie, R.E.; Jiang, M.C.; Rudick, C.N.; Done, J.; Heckman, C.J.; Rosen, J.M.; Schaeffer, A.J.; Klumpp, D.J.
    Acyloxyacyl hydrolase modulates pelvic pain severity (2018), Am. J. Physiol. Regul. Integr. Comp. Physiol., 314, R353-R365 .
    View publication on PubMedView publication on EuropePMC

Application

Application Comment Organism
medicine in a murine neurogenic cystitis model, the gene encoding acyloxyacyl hydrolase is induced in the sacral spinal cord of pseudorabies virus-infected mice. Aoah-deficient mice exhibit increased vesicomotor reflex in response to bladder distension, consistent with spontaneous bladder hypersensitivity, and increased pelvic allodynia in neurogenic cystitis and postbacterial chronic pain models. Aoah deficiency results in greater bladder pathology and tumor necrosis factor production in pseudorabies virus neurogenic cystitis Mus musculus

Organism

Organism UniProt Comment Textmining
Mus musculus O35298
-
-

Synonyms

Synonyms Comment Organism
AOAH
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Mus musculus

General Information

General Information Comment Organism
physiological function in a murine neurogenic cystitis model, the gene encoding acyloxyacyl hydrolase is induced in the sacral spinal cord of pseudorabies virus-infected mice. Aoah-deficient mice exhibit increased vesicomotor reflex in response to bladder distension, consistent with spontaneous bladder hypersensitivity, and increased pelvic allodynia in neurogenic cystitis and postbacterial chronic pain models. Aoah deficiency results in greater bladder pathology and tumor necrosis factor production in pseudorabies virus neurogenic cystitis. Aoah-deficient mice have significantly higher levels of bladder vascular endothelial growth factor Mus musculus