Organism | UniProt | Comment | Textmining |
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Escherichia coli | - |
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General Information | Comment | Organism |
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physiological function | a knockout mutation in alternative sigma factor, rpoS, abolishes putrescine-dependent ygjG-lacZ expression. RpoS overexpression induces ygjG-lacZ expression with putrescine supplementation but not without supplementation. The loss of putrescine-dependent ygjG-lacZ expression induced by rpoS is completely restored under nitrogen-starvation conditions. The putrescine-dependent expression of ygjG-lacZ under this condition is clearly dependent on alternative sigma factor, rpoN, and its cognate activator ntrC | Escherichia coli |