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Literature summary for 2.3.1.5 extracted from
- Dapsone acetylation by human liver arylamine N-acetyltransferases and interaction with antiopportunistic infection drugs (1995), Drug Metab. Dispos., 23, 473-477.
Application
| Application | Comment | Organism |
|---|---|---|
| medicine | inactivation of antiopportunistic infection drugs | Homo sapiens |
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Homo sapiens | - |
- |
- |
Source Tissue
| Source Tissue | Comment | Organism | Textmining |
|---|---|---|---|
| liver | - |
Homo sapiens | - |
Substrates and Products (Substrate)
| Substrates | Comment Substrates | Organism | Products | Comment (Products) | Rev. | Reac. |
|---|---|---|---|---|---|---|
| acetyl-CoA + diaminodiphenylsulfone | i.e. dapsone, predominantly acetylated by NAT2 | Homo sapiens | monoacetyldiaminodiphenylsulfone + CoA | - |
? |  |
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