Disease on EC 2.4.1.155 - alpha-1,6-mannosyl-glycoprotein 6-beta-N-acetylglucosaminyltransferase
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Adenocarcinoma
Clinical and prognostic implications of ?1, 6-N-acetylglucosaminyltransferase V in patients with gastric cancer.
Adenocarcinoma
Knockdown of Mgat5 Inhibits Breast Cancer Cell Growth with Activation of CD4+ T Cells and Macrophages.
Adenocarcinoma
Knockdown of Mgat5 inhibits CD133+ human pulmonary adenocarcinoma cell growth in vitro and in vivo.
Adenocarcinoma of Lung
Altered ?1,6-GlcNAc branched N-glycans impair TGF-?-mediated Epithelial-to-Mesenchymal Transition through Smad signalling pathway in human lung cancer.
Adenocarcinoma of Lung
miR-124-3p Regulates FGF2-EGFR Pathway to Overcome Pemetrexed Resistance in Lung Adenocarcinoma Cells by Targeting MGAT5.
Adenocarcinoma, Mucinous
High expression of N-acetylglucosaminyltransferase V in mucinous tumors of the ovary.
Adenoma
Altered mRNA expression of glycosyltransferases in human colorectal carcinomas and liver metastases.
Adenoma
Co-expression of matriptase and N-acetylglucosaminyltransferase V in thyroid cancer tissues--its possible role in prolonged stability in vivo by aberrant glycosylation.
Adenoma
Post-translational glycoprotein modifications regulate colon cancer stem cells and colon adenoma progression in Apc(min/+) mice through altered Wnt receptor signaling.
alpha-1,6-mannosyl-glycoprotein 6-beta-n-acetylglucosaminyltransferase deficiency
Loss of branched O-mannosyl glycans in astrocytes accelerates remyelination.
alpha-1,6-mannosyl-glycoprotein 6-beta-n-acetylglucosaminyltransferase deficiency
Mgat5 deficiency in T cells and experimental autoimmune encephalomyelitis.
alpha-1,6-mannosyl-glycoprotein 6-beta-n-acetylglucosaminyltransferase deficiency
N-acetylglucosaminyltransferase V (Mgat5)-mediated N-glycosylation negatively regulates Th1 cytokine production by T cells.
alpha-1,6-mannosyl-glycoprotein 6-beta-n-acetylglucosaminyltransferase deficiency
Reactivity of anti-HNK-1 antibodies to branched O-mannose glycans associated with demyelination.
Alzheimer Disease
A secreted type of beta1,6 N-acetylglucosaminyltransferase V (GnT-V), a novel angiogenesis inducer, is regulated by gamma-secretase.
Astrocytoma
Upregulated ?1-6 branch N-glycan marks early gliomagenesis but exhibited biphasic expression in the progression of astrocytic glioma.
Autoimmune Diseases
Genetic Variants of the MGAT5 Gene Are Functionally Implicated in the Modulation of T Cells Glycosylation and Plasma IgG Glycome Composition in Ulcerative Colitis.
Autoimmune Diseases
Negative regulation of T-cell activation and autoimmunity by Mgat5 N-glycosylation.
Brain Injuries
Predominant expression of N-acetylglucosaminyltransferase V (GnT-V) in neural stem/progenitor cells.
Brain Neoplasms
Predictive value of N-acetylglucosaminyltransferase-V for superficial bladder cancer recurrence.
Brain Neoplasms
Radiosensitisation of human glioma cells by inhibition of ?1,6-GlcNAc branched N-glycans.
Breast Neoplasms
?1,6 GlcNAc branches-modified protein tyrosine phosphatase alpha enhances its stability and promotes focal adhesion formation in MCF-7 cells.
Breast Neoplasms
Beta1,6-branched oligosaccharides are increased in lymph node metastases and predict poor outcome in breast carcinoma.
Breast Neoplasms
Chitosan oligosaccharides inhibit epithelial cell migration through blockade of N-acetylglucosaminyltransferase V and branched GlcNAc structure.
Breast Neoplasms
Coexpression of beta1,6-N-acetylglucosaminyltransferase V glycoprotein substrates defines aggressive breast cancers with poor outcome.
Breast Neoplasms
Control of metastasis by Asn-linked, beta1-6 branched oligosaccharides in mouse mammary cancer cells.
Breast Neoplasms
Decreased miR-124-3p promoted breast cancer proliferation and metastasis by targeting MGAT5.
Breast Neoplasms
Inhibition of a specific N-glycosylation activity results in attenuation of breast carcinoma cell invasiveness-related phenotypes: inhibition of epidermal growth factor-induced dephosphorylation of focal adhesion kinase.
Breast Neoplasms
Knockdown of GnT-Va expression inhibits ligand-induced downregulation of the epidermal growth factor receptor and intracellular signaling by inhibiting receptor endocytosis.
Breast Neoplasms
Knockdown of Mgat5 Inhibits Breast Cancer Cell Growth with Activation of CD4+ T Cells and Macrophages.
Breast Neoplasms
Post-translational glycoprotein modifications regulate colon cancer stem cells and colon adenoma progression in Apc(min/+) mice through altered Wnt receptor signaling.
Breast Neoplasms
Specific posttranslational modification regulates early events in mammary carcinoma formation.
Breast Neoplasms
Targeted glycoproteomic identification of biomarkers for human breast carcinoma.
Carcinogenesis
Blocking of N-acetylglucosaminyltransferase V induces cellular endoplasmic reticulum stress in human hepatocarcinoma 7721 cells.
Carcinogenesis
Decreased miR-124-3p promoted breast cancer proliferation and metastasis by targeting MGAT5.
Carcinogenesis
Expression of integrins ?3?1 and ?5?1 and GlcNAc ?1,6 glycan branching influences metastatic melanoma cell migration on fibronectin.
Carcinogenesis
Expression of N-acetylglucosaminyltransferase V in the development of human esophageal cancers: immunohistochemical data from carcinomas and nearby noncancerous lesions.
Carcinogenesis
Hydrogen Sulfide Demonstrates Promising Antitumor Efficacy in Gastric Carcinoma by Targeting MGAT5.
Carcinogenesis
N-Acetylglucosaminyltransferase V exacerbates murine colitis with macrophage dysfunction and enhances colitic tumorigenesis.
Carcinogenesis
N-Acetylglucosaminyltransferase V regulates TGF-? response in hepatic stellate cells and the progression of steatohepatitis.
Carcinogenesis
Positive expressions of N-acetylglucosaminyltransferase-V (GnT-V) and beta1-6 branching N-linked oligosaccharides in human testicular germ cells diminish during malignant transformation and progression.
Carcinogenesis
Specific posttranslational modification regulates early events in mammary carcinoma formation.
Carcinogenesis
Targeted glycoproteomic identification of biomarkers for human breast carcinoma.
Carcinogenesis
Transcriptional regulation of the protocadherin ? cluster during Her-2 protein-induced mammary tumorigenesis results from altered N-glycan branching.
Carcinoma
?1,6-N-acetylglucosaminyltransferase V predicts recurrence and survival of patients with clear-cell renal cell carcinoma after surgical resection.
Carcinoma
Altered mRNA expression of glycosyltransferases in human colorectal carcinomas and liver metastases.
Carcinoma
Beta1,6-branched oligosaccharides and coarse vesicles: a common, pervasive phenotype in melanoma and other human cancers.
Carcinoma
Chemical enhancers of cytokine signaling that suppress microfilament turnover and tumor cell growth.
Carcinoma
Co-expression of matriptase and N-acetylglucosaminyltransferase V in thyroid cancer tissues--its possible role in prolonged stability in vivo by aberrant glycosylation.
Carcinoma
Deletion of mouse embryo fibroblast N-acetylglucosaminyltransferase V stimulates alpha5beta1 integrin expression mediated by the protein kinase C signaling pathway.
Carcinoma
Effect of GnT-V knockdown on the proliferation, migration and invasion of the SMMC7721/R human hepatocellular carcinoma drug-resistant cell line.
Carcinoma
Expression of N-acetylglucosaminyltransferase V in gastric cancer correlates with metastasis and prognosis.
Carcinoma
Expression of N-acetylglucosaminyltransferase V in the development of human esophageal cancers: immunohistochemical data from carcinomas and nearby noncancerous lesions.
Carcinoma
Expression of N-acetylglucosaminyltransferase V in the subserosal layer correlates with postsurgical survival of pathological tumor stage 2 carcinoma of the gallbladder.
Carcinoma
Expression of N-acetylglucosaminyltransferase v is associated with prognosis and histology in non-small cell lung cancers.
Carcinoma
Hydrogen Sulfide Demonstrates Promising Antitumor Efficacy in Gastric Carcinoma by Targeting MGAT5.
Carcinoma
Inhibition of a specific N-glycosylation activity results in attenuation of breast carcinoma cell invasiveness-related phenotypes: inhibition of epidermal growth factor-induced dephosphorylation of focal adhesion kinase.
Carcinoma
Knockdown of GnT-Va expression inhibits ligand-induced downregulation of the epidermal growth factor receptor and intracellular signaling by inhibiting receptor endocytosis.
Carcinoma
Negative expression of N-acetylglucosaminyltransferase V in oral squamous cell carcinoma correlates with poor prognosis.
Carcinoma
Transcriptional regulation of the N-acetylglucosaminyltransferase V gene in human bile duct carcinoma cells (HuCC-T1) is mediated by Ets-1.
Carcinoma, Hepatocellular
Characterization of UDP-N-acetylglucosamine:alpha-6-d-mannoside beta-1,6-N-acetylglucosaminyltransferase V from a human hepatoma cell line Hep3B.
Carcinoma, Hepatocellular
Determination of N-acetylglucosaminyltransferases III, IV and V in normal and hepatoma tissues of rats.
Carcinoma, Hepatocellular
Effect of N-acetylglucosaminyltransferase V on the expressions of other glycosyltransferases.
Carcinoma, Hepatocellular
Effects of epidermal growth factor and insulin on the activity of N-acetylglucosaminyltransferase V.
Carcinoma, Hepatocellular
EGF-mediated migration signaling activated by N-acetylglucosaminyltransferase-V via receptor protein tyrosine phosphatase kappa.
Carcinoma, Hepatocellular
Elevated activity of N-acetylglucosaminyltransferase V in human hepatocellular carcinoma.
Carcinoma, Hepatocellular
Elevated expression of UDP-N-acetylglucosamine: alphamannoside beta1,6 N-acetylglucosaminyltransferase is an early event in hepatocarcinogenesis.
Carcinoma, Hepatocellular
Hepatic aberrant glycosylation by N-acetylglucosaminyltransferase V accelerates HDL assembly.
Carcinoma, Hepatocellular
Increase in beta1-6 GlcNAc branching caused by N-acetylglucosaminyltransferase V directs integrin beta1 stability in human hepatocellular carcinoma cell line SMMC-7721.
Carcinoma, Hepatocellular
Increased expression of N-acetylglucosaminyltransferase-V in human hepatoma cells by retinoic acid and 1alpha,25-dihydroxyvitamin D3.
Carcinoma, Hepatocellular
Loss of Barx1 promotes hepatocellular carcinoma metastasis through up-regulating MGAT5 and MMP9 expression and indicates poor prognosis.
Carcinoma, Hepatocellular
N-acetylglucosaminyltransferase III and V messenger RNA levels in LEC rats during hepatocarcinogenesis.
Carcinoma, Hepatocellular
N-acetylglucosaminyltransferase V activity in metastatic models of human hepatocellular carcinoma in nude mice.
Carcinoma, Hepatocellular
N-Acetylglucosaminyltransferase V as a possible aid for the evaluation of tumor invasiveness in patients with hepatocellular carcinoma.
Carcinoma, Hepatocellular
N-acetylglucosaminyltransferase V confers hepatoma cells with resistance to anoikis through EGFR/PAK1 activation.
Carcinoma, Hepatocellular
Predictive value of N-acetylglucosaminyltransferase-V for superficial bladder cancer recurrence.
Carcinoma, Hepatocellular
Prognostic significance of ?1,6-N-acetylglucosaminyltransferase V expression in patients with hepatocellular carcinoma.
Carcinoma, Hepatocellular
The effect of receptor protein tyrosine phosphatase kappa on the change of cell adhesion and proliferation induced by N-acetylglucosaminyltransferase V.
Carcinoma, Hepatocellular
The role of N-acetylglucosaminyltransferases V in the malignancy of human hepatocellular carcinoma.
Carcinoma, Ovarian Epithelial
High expression of N-acetylglucosaminyltransferase V in mucinous tumors of the ovary.
Carcinoma, Papillary
Co-expression of matriptase and N-acetylglucosaminyltransferase V in thyroid cancer tissues--its possible role in prolonged stability in vivo by aberrant glycosylation.
Carcinoma, Renal Cell
?1,6-N-acetylglucosaminyltransferase V predicts recurrence and survival of patients with clear-cell renal cell carcinoma after surgical resection.
Carcinoma, Squamous Cell
Expression of N-acetylglucosaminyltransferase V in the development of human esophageal cancers: immunohistochemical data from carcinomas and nearby noncancerous lesions.
Carcinoma, Squamous Cell
Expression of N-acetylglucosaminyltransferase v is associated with prognosis and histology in non-small cell lung cancers.
Choriocarcinoma
N-acetylglucosaminyltransferase V regulates extravillous trophoblast invasion through glycosylation of alpha5beta1 integrin.
Colitis
N-Acetylglucosaminyltransferase V exacerbates murine colitis with macrophage dysfunction and enhances colitic tumorigenesis.
Colitis, Ulcerative
Genetic Variants of the MGAT5 Gene Are Functionally Implicated in the Modulation of T Cells Glycosylation and Plasma IgG Glycome Composition in Ulcerative Colitis.
Colonic Neoplasms
Attachment of human colon cancer cells to vascular endothelium is enhanced by N-acetylglucosaminyltransferase V.
Colonic Neoplasms
Comparative quantitation of aberrant glycoforms by lectin-based glycoprotein enrichment coupled with multiple-reaction monitoring mass spectrometry.
Colonic Neoplasms
Functional proteomics study reveals that N-Acetylglucosaminyltransferase V reinforces the invasive/metastatic potential of colon cancer through aberrant glycosylation on tissue inhibitor of metalloproteinase-1.
Colonic Neoplasms
Lectin from Sambucus sieboldiana abrogates the anoikis resistance of colon cancer cells conferred by N-acetylglucosaminyltransferase V during hematogenous metastasis.
Colonic Neoplasms
Post-translational glycoprotein modifications regulate colon cancer stem cells and colon adenoma progression in Apc(min/+) mice through altered Wnt receptor signaling.
Colonic Neoplasms
Predictive value of N-acetylglucosaminyltransferase-V for superficial bladder cancer recurrence.
Colorectal Neoplasms
Altered mRNA expression of glycosyltransferases in human colorectal carcinomas and liver metastases.
Colorectal Neoplasms
Expression of N-acetylglucosaminyltransferase V in colorectal cancer correlates with metastasis and poor prognosis.
Colorectal Neoplasms
Quantitative analysis of an aberrant glycoform of TIMP1 from colon cancer serum by L-PHA-enrichment and SISCAPA with MRM mass spectrometry.
Colorectal Neoplasms
Silencing GnT-V reduces oxaliplatin chemosensitivity in human colorectal cancer cells through N-glycan alteration of organic cation transporter member 2.
Demyelinating Diseases
Genetics and the environment converge to dysregulate N-glycosylation in multiple sclerosis.
Demyelinating Diseases
Mgat5 deficiency in T cells and experimental autoimmune encephalomyelitis.
Demyelinating Diseases
Reactivity of anti-HNK-1 antibodies to branched O-mannose glycans associated with demyelination.
Diabetes Mellitus, Type 1
Incorporating parental information into family-based association tests.
Encephalomyelitis
Mgat5 deficiency in T cells and experimental autoimmune encephalomyelitis.
Encephalomyelitis, Autoimmune, Experimental
Mgat5 deficiency in T cells and experimental autoimmune encephalomyelitis.
Endometrial Neoplasms
Expression of N-acetylglucosaminyltransferase V in endometrial cancer correlates with poor prognosis.
Esophageal Neoplasms
Expression of N-acetylglucosaminyltransferase V in the development of human esophageal cancers: immunohistochemical data from carcinomas and nearby noncancerous lesions.
Fatty Liver
N-Acetylglucosaminyltransferase V regulates TGF-? response in hepatic stellate cells and the progression of steatohepatitis.
Fibrosarcoma
Aberrant N-glycosylation of beta1 integrin causes reduced alpha5beta1 integrin clustering and stimulates cell migration.
Fibrosarcoma
Knockdown of GnT-Va expression inhibits ligand-induced downregulation of the epidermal growth factor receptor and intracellular signaling by inhibiting receptor endocytosis.
Fibrosarcoma
N-acetylglucosaminyltransferase V expression levels regulate cadherin-associated homotypic cell-cell adhesion and intracellular signaling pathways.
Fibrosarcoma
Regulation of homotypic cell-cell adhesion by branched N-glycosylation of N-cadherin extracellular EC2-3 domains.
Glioblastoma
Phostine PST3.1a Targets MGAT5 and Inhibits Glioblastoma-Initiating Cell Invasiveness and Proliferation.
Glioblastoma
Upregulated ?1-6 branch N-glycan marks early gliomagenesis but exhibited biphasic expression in the progression of astrocytic glioma.
Glioma
?1,6 GlcNAc branches-modified protein tyrosine phosphatase Mu attenuates its tyrosine phosphatase activity and promotes glioma cell migration through PLC?-PKC pathways.
Glioma
Antigenic profiling of glioma cells to generate allogeneic vaccines or dendritic cell-based therapeutics.
Glioma
Beta1,6-N-acetylglucosamine-bearing N-glycans in human gliomas: implications for a role in regulating invasivity.
Glioma
Glioma stem cells invasive phenotype at optimal stiffness is driven by MGAT5 dependent mechanosensing.
Glioma
Radiosensitisation of human glioma cells by inhibition of ?1,6-GlcNAc branched N-glycans.
Glioma
Terminally sialylated and fucosylated complex N-glycans are involved in the malignant behavior of high-grade glioma.
Glioma
Upregulated ?1-6 branch N-glycan marks early gliomagenesis but exhibited biphasic expression in the progression of astrocytic glioma.
Hepatitis
N-acetylglucosaminyltransferase III and V messenger RNA levels in LEC rats during hepatocarcinogenesis.
Hepatitis, Chronic
Elevated expression of UDP-N-acetylglucosamine: alphamannoside beta1,6 N-acetylglucosaminyltransferase is an early event in hepatocarcinogenesis.
Hepatitis, Chronic
N-Acetylglucosaminyltransferase V regulates TGF-? response in hepatic stellate cells and the progression of steatohepatitis.
Infections
?1,6 GlcNAc branches-modified protein tyrosine phosphatase Mu attenuates its tyrosine phosphatase activity and promotes glioma cell migration through PLC?-PKC pathways.
Infections
N-acetylglucosaminyltransferase V-deficiency increases susceptibility to murine malaria.
Liver Cirrhosis
Elevated expression of UDP-N-acetylglucosamine: alphamannoside beta1,6 N-acetylglucosaminyltransferase is an early event in hepatocarcinogenesis.
Liver Cirrhosis
Knockdown of N-acetylglucosaminyl transferase v ameliorates hepatotoxin-induced liver fibrosis in mice.
Liver Cirrhosis
Oligosaccharide modification by N-acetylglucosaminyltransferase-V in macrophages are involved in pathogenesis of bleomycin-induced scleroderma.
Liver Cirrhosis
The role of N-acetylglucosaminyltransferases V in the malignancy of human hepatocellular carcinoma.
Liver Diseases
N-Acetylglucosaminyltransferase V regulates TGF-? response in hepatic stellate cells and the progression of steatohepatitis.
Liver Neoplasms
IGF2BP1 promotes the liver cancer stem cell phenotype by regulating MGAT5 mRNA stability via m6A RNA methylation.
Liver Neoplasms
Prognostic significance of ?1,6-N-acetylglucosaminyltransferase V expression in patients with hepatocellular carcinoma.
Lung Neoplasms
Altered ?1,6-GlcNAc branched N-glycans impair TGF-?-mediated Epithelial-to-Mesenchymal Transition through Smad signalling pathway in human lung cancer.
Lung Neoplasms
Down-regulation of N-acetylglucosaminyltransferase V by tumorigenesis- or metastasis-suppressor gene and its relation to metastatic potential of human hepatocarcinoma cells.
Lung Neoplasms
Expression of N-acetylglucosaminyltransferase v is associated with prognosis and histology in non-small cell lung cancers.
Lung Neoplasms
High Throughput Multiplex SNP-analysis in Chronic Obstructive Pulmonary Disease and Lung Cancer.
Lung Neoplasms
N-acetylglucosaminyltransferase V modulates radiosensitivity and migration of small cell lung cancer through epithelial-mesenchymal transition.
Lung Neoplasms
Purification and characterization of UDP-N-acetylglucosamine: alpha-6-D-mannoside beta 1-6N-acetylglucosaminyltransferase (N-acetylglucosaminyltransferase V) from a human lung cancer cell line.
Lymphatic Metastasis
Expression of N-acetylglucosaminyltransferase V in the development of human esophageal cancers: immunohistochemical data from carcinomas and nearby noncancerous lesions.
Lymphatic Metastasis
N-acetylglucosaminyltransferase III and V messenger RNA levels in LEC rats during hepatocarcinogenesis.
Lymphatic Metastasis
The implication of N-acetylglucosaminyltransferase V expression in gastric cancer.
Lymphoma
Alpha-2,6-sialylation of L-PHA reactive oligosaccharides and expression of N-acetylglucosaminyltransferase V in human diffuse large B cell lymphoma.
Lymphoma, B-Cell
Alpha-2,6-sialylation of L-PHA reactive oligosaccharides and expression of N-acetylglucosaminyltransferase V in human diffuse large B cell lymphoma.
Lymphoma, Large B-Cell, Diffuse
Alpha-2,6-sialylation of L-PHA reactive oligosaccharides and expression of N-acetylglucosaminyltransferase V in human diffuse large B cell lymphoma.
Malaria
N-acetylglucosaminyltransferase V-deficiency increases susceptibility to murine malaria.
Melanoma
A peptide recognized by human cytolytic T lymphocytes on HLA-A2 melanomas is encoded by an intron sequence of the N-acetylglucosaminyltransferase V gene.
Melanoma
Beta1,6-branched oligosaccharides regulate melanin content and motility in macrophage-melanoma fusion hybrids.
Melanoma
Co-localization of beta1,6-branched oligosaccharides and coarse melanin in macrophage-melanoma fusion hybrids and human melanoma cells in vitro.
Melanoma
Diverse expression of N-acetylglucosaminyltransferase V and complex-type ?1,6-branched N-glycans in uveal and cutaneous melanoma cells.
Melanoma
Expression of c-Met proto-oncogene in metastatic macrophage x melanoma fusion hybrids: implication of its possible role in MSH-induced motility.
Melanoma
Fusion hybrids with macrophage and melanoma cells up-regulate N-acetylglucosaminyltransferase V, beta1-6 branching, and metastasis.
Melanoma
GnT-V expression and metastatic phenotypes in macrophage-melanoma fusion hybrids is down-regulated by 5-Aza-dC: evidence for methylation sensitive, extragenic regulation of GnT-V transcription.
Melanoma
Implication of N-acetylglucosaminyltransferases III and V in cancer: gene regulation and signaling mechanism.
Melanoma
Membrane protein regulators of melanoma pulmonary colonisation identified using a CRISPRa screen and spontaneous metastasis assay in mice.
Melanoma
Overexpression of N-acetylglucosaminyltransferases III and V in human melanoma cells. Implications for MCAM N-glycosylation.
Melanoma
Transforming growth factor beta up-regulates expression of the N-acetylglucosaminyltransferase V gene in mouse melanoma cells.
Melanoma
Upregulation of alpha and beta integrin subunits in metastatic macrophage-melanoma fusion hybrids.
Melanoma, Experimental
Co-localization of beta1,6-branched oligosaccharides and coarse melanin in macrophage-melanoma fusion hybrids and human melanoma cells in vitro.
Melanoma, Experimental
Expression of c-Met proto-oncogene in metastatic macrophage x melanoma fusion hybrids: implication of its possible role in MSH-induced motility.
Melanoma, Experimental
UDP-GlcNAc concentration is an important factor in the biosynthesis of beta1,6-branched oligosaccharides: regulation based on the kinetic properties of N-acetylglucosaminyltransferase V.
Mouth Neoplasms
Negative expression of N-acetylglucosaminyltransferase V in oral squamous cell carcinoma correlates with poor prognosis.
Multiple Sclerosis
Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants.
Multiple Sclerosis
Negative regulation of T-cell activation and autoimmunity by Mgat5 N-glycosylation.
Multiple Sclerosis
Oligosaccharide modification by N-acetylglucosaminyltransferase-V in macrophages are involved in pathogenesis of bleomycin-induced scleroderma.
Nasopharyngeal Carcinoma
Down-regulation of GnT-V enhances nasopharyngeal carcinoma cell CNE-2 radiosensitivity in vitro and in vivo.
Nasopharyngeal Carcinoma
Down-regulation of GnT-V inhibits nasopharyngeal carcinoma cell CNE-2 malignancy in vitro and in vivo.
Nasopharyngeal Carcinoma
Inhibition of N-acetylglucosaminyltransferase V enhances the cetuximab-induced radiosensitivity of nasopharyngeal carcinoma cells likely through EGFR N-glycan alterations.
Nasopharyngeal Carcinoma
Reversal effect of GnT-V on the radioresistance of human nasopharyngeal carcinoma cells by alteration ?1, 6-GlcNAc branched N-glycans.
Neoplasm Metastasis
A secreted type of beta 1,6-N-acetylglucosaminyltransferase V (GnT-V) induces tumor angiogenesis without mediation of glycosylation: a novel function of GnT-V distinct from the original glycosyltransferase activity.
Neoplasm Metastasis
A secreted type of beta1,6 N-acetylglucosaminyltransferase V (GnT-V), a novel angiogenesis inducer, is regulated by gamma-secretase.
Neoplasm Metastasis
Addition of beta1-6 GlcNAc branching to the oligosaccharide attached to Asn 772 in the serine protease domain of matriptase plays a pivotal role in its stability and resistance against trypsin.
Neoplasm Metastasis
Altered ?1,6-GlcNAc and bisecting GlcNAc-branched N-glycan on integrin ?1 are associated with early spontaneous miscarriage in humans.
Neoplasm Metastasis
Altered mRNA expression of glycosyltransferases in human colorectal carcinomas and liver metastases.
Neoplasm Metastasis
Attachment of human colon cancer cells to vascular endothelium is enhanced by N-acetylglucosaminyltransferase V.
Neoplasm Metastasis
Beta1,6-branched oligosaccharides and coarse vesicles: a common, pervasive phenotype in melanoma and other human cancers.
Neoplasm Metastasis
Beta1,6-branched oligosaccharides are increased in lymph node metastases and predict poor outcome in breast carcinoma.
Neoplasm Metastasis
Coexpression of beta1,6-N-acetylglucosaminyltransferase V glycoprotein substrates defines aggressive breast cancers with poor outcome.
Neoplasm Metastasis
Correlation of glycosyltransferases mRNA expression in extrahepatic bile duct carcinoma with clinical pathological characteristics.
Neoplasm Metastasis
Decreased miR-124-3p promoted breast cancer proliferation and metastasis by targeting MGAT5.
Neoplasm Metastasis
Down-regulation of N-acetylglucosaminyltransferase V by tumorigenesis- or metastasis-suppressor gene and its relation to metastatic potential of human hepatocarcinoma cells.
Neoplasm Metastasis
Downregulation of the GnT-V gene inhibits metastasis and invasion of BGC823 gastric cancer cells.
Neoplasm Metastasis
Elevated expression of N-acetylglucosaminyltransferase V in first trimester human placenta.
Neoplasm Metastasis
Evaluating the function of matriptase and N-acetylglucosaminyltransferase V in prostate cancer metastasis.
Neoplasm Metastasis
Expression of N-acetylglucosaminyltransferase V in colorectal cancer correlates with metastasis and poor prognosis.
Neoplasm Metastasis
Expression of N-acetylglucosaminyltransferase V in gastric cancer correlates with metastasis and prognosis.
Neoplasm Metastasis
Expression of N-acetylglucosaminyltransferase V in the development of human esophageal cancers: immunohistochemical data from carcinomas and nearby noncancerous lesions.
Neoplasm Metastasis
Expression of N-acetylglucosaminyltransferase V in the subserosal layer correlates with postsurgical survival of pathological tumor stage 2 carcinoma of the gallbladder.
Neoplasm Metastasis
Expression of N-acetylglucosaminyltransferase v is associated with prognosis and histology in non-small cell lung cancers.
Neoplasm Metastasis
Fusion hybrids with macrophage and melanoma cells up-regulate N-acetylglucosaminyltransferase V, beta1-6 branching, and metastasis.
Neoplasm Metastasis
Glycans and cancer: role of N-glycans in cancer biomarker, progression and metastasis, and therapeutics.
Neoplasm Metastasis
GnT-V expression and metastatic phenotypes in macrophage-melanoma fusion hybrids is down-regulated by 5-Aza-dC: evidence for methylation sensitive, extragenic regulation of GnT-V transcription.
Neoplasm Metastasis
Hydrogen Sulfide Demonstrates Promising Antitumor Efficacy in Gastric Carcinoma by Targeting MGAT5.
Neoplasm Metastasis
Identification of liver metastasis-related genes in a novel human pancreatic carcinoma cell model by microarray analysis.
Neoplasm Metastasis
Implication of GnT-V in cancer metastasis: a glycomic approach for identification of a target protein and its unique function as an angiogenic cofactor.
Neoplasm Metastasis
Implication of N-acetylglucosaminyltransferases III and V in cancer: gene regulation and signaling mechanism.
Neoplasm Metastasis
Knockdown of Mgat5 Inhibits Breast Cancer Cell Growth with Activation of CD4+ T Cells and Macrophages.
Neoplasm Metastasis
Lectin from Sambucus sieboldiana abrogates the anoikis resistance of colon cancer cells conferred by N-acetylglucosaminyltransferase V during hematogenous metastasis.
Neoplasm Metastasis
Loss of Barx1 promotes hepatocellular carcinoma metastasis through up-regulating MGAT5 and MMP9 expression and indicates poor prognosis.
Neoplasm Metastasis
Membrane protein regulators of melanoma pulmonary colonisation identified using a CRISPRa screen and spontaneous metastasis assay in mice.
Neoplasm Metastasis
Metabolic homeostasis and tissue renewal are dependent on beta1,6GlcNAc-branched N-glycans.
Neoplasm Metastasis
Modulation of CD147-induced matrix metalloproteinase activity: role of CD147 N-glycosylation.
Neoplasm Metastasis
N-acetylglucosaminyltransferase III and V messenger RNA levels in LEC rats during hepatocarcinogenesis.
Neoplasm Metastasis
N-acetylglucosaminyltransferase III antagonizes the effect of N-acetylglucosaminyltransferase V on alpha3beta1 integrin-mediated cell migration.
Neoplasm Metastasis
N-acetylglucosaminyltransferase V inhibits the invasion of trophoblast cells by attenuating MMP2/9 activity in early human pregnancy.
Neoplasm Metastasis
N-Acetylglucosaminyltransferase V regulates TGF-? response in hepatic stellate cells and the progression of steatohepatitis.
Neoplasm Metastasis
N-glycosylation by N-acetylglucosaminyltransferase V enhances the interaction of CD147/basigin with integrin ?1 and promotes HCC metastasis.
Neoplasm Metastasis
Negative expression of N-acetylglucosaminyltransferase V in oral squamous cell carcinoma correlates with poor prognosis.
Neoplasm Metastasis
NMR structural characterization of substrates bound to N-acetylglucosaminyltransferase V.
Neoplasm Metastasis
Oligosaccharide modification by N-acetylglucosaminyltransferase-V in macrophages are involved in pathogenesis of bleomycin-induced scleroderma.
Neoplasm Metastasis
Physiological roles of N-acetylglucosaminyltransferase V(GnT-V) in mice.
Neoplasm Metastasis
Prometastatic effect of N-acetylglucosaminyltransferase V is due to modification and stabilization of active matriptase by adding beta 1-6 GlcNAc branching.
Neoplasm Metastasis
Regulation of cytokine receptors by Golgi N-glycan processing and endocytosis.
Neoplasm Metastasis
Regulation of the GnT-V promoter by transcription factor Ets-1 in various cancer cell lines.
Neoplasm Metastasis
Relationship between metastasis-associated phenotypes and N-glycan structure of surface glycoproteins in human hepatocarcinoma cells.
Neoplasm Metastasis
Sequences of the mouse N-acetylglucosaminyltransferase V (Mgat5) mRNA and an mRNA expressed by an Mgat5-deficient cell line.
Neoplasm Metastasis
Structure and mechanism of cancer-associated N-acetylglucosaminyltransferase-V.
Neoplasm Metastasis
Suppression of tumor-related glycosylation of cell surface receptors by the 16-kDa membrane subunit of vacuolar H+-ATPase.
Neoplasm Metastasis
Synthesis and evaluation of a radioiodinated trisaccharide derivative as a synthetic substrate for a sensitive N-acetylglucosaminyltransferase V radioassay.
Neoplasm Metastasis
TGF-?1 Causes EMT by regulating N-Acetyl Glucosaminyl Transferases via Downregulation of Non Muscle Myosin II-A through JNK/P38/PI3K pathway in lung cancer.
Neoplasm Metastasis
The Caenorhabditis elegans gene, gly-2, can rescue the N-acetylglucosaminyltransferase V mutation of Lec4 cells.
Neoplasm Metastasis
The implication of N-acetylglucosaminyltransferase V expression in gastric cancer.
Neoplasm Metastasis
The Role of N-acetylglucosaminyltransferase III and V in the Post-Transcriptional Modifications of E-cadherin.
Neoplasm Metastasis
Transforming growth factor beta up-regulates expression of the N-acetylglucosaminyltransferase V gene in mouse melanoma cells.
Neoplasm Metastasis
True significance of N-acetylglucosaminyltransferases GnT-III, V and ?1,6 fucosyltransferase in epithelial-mesenchymal transition and cancer.
Neoplasm Metastasis
Upregulation of N-acetylglucosaminyltransferase-V by heparin-binding EGF-like growth factor induces keratinocyte proliferation and epidermal hyperplasia.
Neoplasm Metastasis
[GnT-V overexpression in human hepatocarcinoma cells affects its migration and expression of cell adhesion molecules]
Neoplasms
A genetic strategy involving a glycosyltransferase promoter and a lipid translocating enzyme to eliminate cancer cells.
Neoplasms
A secreted type of beta 1,6-N-acetylglucosaminyltransferase V (GnT-V) induces tumor angiogenesis without mediation of glycosylation: a novel function of GnT-V distinct from the original glycosyltransferase activity.
Neoplasms
A secreted type of beta1,6 N-acetylglucosaminyltransferase V (GnT-V), a novel angiogenesis inducer, is regulated by gamma-secretase.
Neoplasms
Aberrant N-glycosylation of beta1 integrin causes reduced alpha5beta1 integrin clustering and stimulates cell migration.
Neoplasms
Addition of beta1-6 GlcNAc branching to the oligosaccharide attached to Asn 772 in the serine protease domain of matriptase plays a pivotal role in its stability and resistance against trypsin.
Neoplasms
Alteration in N -acetylglucosaminyltransferase activities and glycan structure in tissue and bile glycoproteins from extrahepatic bile duct carcinoma.
Neoplasms
Altered ?1,6-GlcNAc branched N-glycans impair TGF-?-mediated Epithelial-to-Mesenchymal Transition through Smad signalling pathway in human lung cancer.
Neoplasms
Attachment of human colon cancer cells to vascular endothelium is enhanced by N-acetylglucosaminyltransferase V.
Neoplasms
Beta1,6-branched oligosaccharides and coarse vesicles: a common, pervasive phenotype in melanoma and other human cancers.
Neoplasms
Beta1,6-branched oligosaccharides are increased in lymph node metastases and predict poor outcome in breast carcinoma.
Neoplasms
Caveolin-1 regulation of dynamin-dependent, raft-mediated endocytosis of cholera toxin b-subunit occurs independently of caveolae.
Neoplasms
Co-expression of matriptase and N-acetylglucosaminyltransferase V in thyroid cancer tissues--its possible role in prolonged stability in vivo by aberrant glycosylation.
Neoplasms
Coexpression of beta1,6-N-acetylglucosaminyltransferase V glycoprotein substrates defines aggressive breast cancers with poor outcome.
Neoplasms
Correlated gene expression between beta-1,4-galactosyltransferase V and N-acetylglucosaminyltransferase V in human cancer cell lines.
Neoplasms
Correlation of glycosyltransferases mRNA expression in extrahepatic bile duct carcinoma with clinical pathological characteristics.
Neoplasms
Deletion of mouse embryo fibroblast N-acetylglucosaminyltransferase V stimulates alpha5beta1 integrin expression mediated by the protein kinase C signaling pathway.
Neoplasms
Down-regulation of GnT-V inhibits nasopharyngeal carcinoma cell CNE-2 malignancy in vitro and in vivo.
Neoplasms
Down-regulation of N-acetylglucosaminyltransferase V by tumorigenesis- or metastasis-suppressor gene and its relation to metastatic potential of human hepatocarcinoma cells.
Neoplasms
Down-regulation of N-acetylglucosaminyltransferase-V induces ER stress by changing glycosylation and function of GLUT1.
Neoplasms
E-cadherin and adherens-junctions stability in gastric carcinoma: Functional implications of glycosyltransferases involving N-glycan branching biosynthesis, N-acetylglucosaminyltransferases III and V.
Neoplasms
Ectopic expression of N-acetylglucosaminyltransferase V accelerates hepatic triglyceride synthesis.
Neoplasms
EGF-mediated migration signaling activated by N-acetylglucosaminyltransferase-V via receptor protein tyrosine phosphatase kappa.
Neoplasms
Elevated expression of N-acetylglucosaminyltransferase V in first trimester human placenta.
Neoplasms
Enhanced Epithelial-Mesenchymal Transition-like Phenotype in N-Acetylglucosaminyltransferase V Transgenic Mouse Skin Promotes Wound Healing.
Neoplasms
Expression of integrins ?3?1 and ?5?1 and GlcNAc ?1,6 glycan branching influences metastatic melanoma cell migration on fibronectin.
Neoplasms
Expression of N-acetylglucosaminyltransferase V in colorectal cancer correlates with metastasis and poor prognosis.
Neoplasms
Expression of N-acetylglucosaminyltransferase V in endometrial cancer correlates with poor prognosis.
Neoplasms
Expression of N-acetylglucosaminyltransferase V in gastric cancer correlates with metastasis and prognosis.
Neoplasms
Expression of N-acetylglucosaminyltransferase V in the development of human esophageal cancers: immunohistochemical data from carcinomas and nearby noncancerous lesions.
Neoplasms
Expression of N-acetylglucosaminyltransferase V in the subserosal layer correlates with postsurgical survival of pathological tumor stage 2 carcinoma of the gallbladder.
Neoplasms
Expression of N-acetylglucosaminyltransferase v is associated with prognosis and histology in non-small cell lung cancers.
Neoplasms
Functional proteomics study reveals that N-Acetylglucosaminyltransferase V reinforces the invasive/metastatic potential of colon cancer through aberrant glycosylation on tissue inhibitor of metalloproteinase-1.
Neoplasms
Fusion hybrids with macrophage and melanoma cells up-regulate N-acetylglucosaminyltransferase V, beta1-6 branching, and metastasis.
Neoplasms
Generation of antibodies recognizing an aberrant glycoform of human tissue inhibitor of metalloproteinase-1 (TIMP-1) using decoy immunization and phage display.
Neoplasms
Glioma stem cells invasive phenotype at optimal stiffness is driven by MGAT5 dependent mechanosensing.
Neoplasms
Glycans and cancer: role of N-glycans in cancer biomarker, progression and metastasis, and therapeutics.
Neoplasms
GnT-V expression and metastatic phenotypes in macrophage-melanoma fusion hybrids is down-regulated by 5-Aza-dC: evidence for methylation sensitive, extragenic regulation of GnT-V transcription.
Neoplasms
GnT-V promotes chemosensitivity to gemcitabine in bladder cancer cells through ?1,6 GlcNAc branch modification of human equilibrative nucleoside transporter 1.
Neoplasms
Hepatic aberrant glycosylation by N-acetylglucosaminyltransferase V accelerates HDL assembly.
Neoplasms
High expression of N-acetylglucosaminyltransferase V in mucinous tumors of the ovary.
Neoplasms
Identification of liver metastasis-related genes in a novel human pancreatic carcinoma cell model by microarray analysis.
Neoplasms
Implication of GnT-V in cancer metastasis: a glycomic approach for identification of a target protein and its unique function as an angiogenic cofactor.
Neoplasms
Implication of N-acetylglucosaminyltransferases III and V in cancer: gene regulation and signaling mechanism.
Neoplasms
In vivo delivery of bio-nanocapsules displaying L4-PHA isolectin to malignant tumors overexpressing N-acetylglucosaminyltransferase V.
Neoplasms
In vivo delivery of bionanocapsules displaying Phaseolus vulgaris agglutinin-L4 isolectin to malignant tumors overexpressing N-acetylglucosaminyltransferase V.
Neoplasms
Inhibition of a specific N-glycosylation activity results in attenuation of breast carcinoma cell invasiveness-related phenotypes: inhibition of epidermal growth factor-induced dephosphorylation of focal adhesion kinase.
Neoplasms
Inhibition of N-acetylglucosaminyltransferase V enhances sensitivity of radiotherapy in human prostate cancer.
Neoplasms
Inhibition of N-acetylglucosaminyltransferase V enhances the cetuximab-induced radiosensitivity of nasopharyngeal carcinoma cells likely through EGFR N-glycan alterations.
Neoplasms
Knockdown of Mgat5 Inhibits Breast Cancer Cell Growth with Activation of CD4+ T Cells and Macrophages.
Neoplasms
Knockdown of Mgat5 inhibits CD133+ human pulmonary adenocarcinoma cell growth in vitro and in vivo.
Neoplasms
Lectin from Sambucus sieboldiana abrogates the anoikis resistance of colon cancer cells conferred by N-acetylglucosaminyltransferase V during hematogenous metastasis.
Neoplasms
Lectin precipitation using phytohemagglutinin-L(4) coupled to avidin-agarose for serological biomarker discovery in colorectal cancer.
Neoplasms
Loss of expression of N-acetylglucosaminyltransferase Va results in altered gene expression of glycosyltransferases and galectins.
Neoplasms
Membrane protein regulators of melanoma pulmonary colonisation identified using a CRISPRa screen and spontaneous metastasis assay in mice.
Neoplasms
Metabolic homeostasis and tissue renewal are dependent on beta1,6GlcNAc-branched N-glycans.
Neoplasms
Mevalonate Pathway Inhibition Slows Breast Cancer Metastasis via Reduced N-glycosylation Abundance and Branching.
Neoplasms
Mgat5 modulates the effect of early life stress on adult behavior and physical health in mice.
Neoplasms
miR-124-3p Regulates FGF2-EGFR Pathway to Overcome Pemetrexed Resistance in Lung Adenocarcinoma Cells by Targeting MGAT5.
Neoplasms
Modulation of CD147-induced matrix metalloproteinase activity: role of CD147 N-glycosylation.
Neoplasms
N-acetylglucosaminyltransferase III and V messenger RNA levels in LEC rats during hepatocarcinogenesis.
Neoplasms
N-acetylglucosaminyltransferase V and beta1-6 branching N-linked oligosaccharides are associated with good prognosis of patients with bladder cancer.
Neoplasms
N-Acetylglucosaminyltransferase V as a possible aid for the evaluation of tumor invasiveness in patients with hepatocellular carcinoma.
Neoplasms
N-acetylglucosaminyltransferase V confers hepatoma cells with resistance to anoikis through EGFR/PAK1 activation.
Neoplasms
N-acetylglucosaminyltransferase V expression levels regulate cadherin-associated homotypic cell-cell adhesion and intracellular signaling pathways.
Neoplasms
N-acetylglucosaminyltransferase V inhibits the invasion of trophoblast cells by attenuating MMP2/9 activity in early human pregnancy.
Neoplasms
N-acetylglucosaminyltransferase V mediates cell migration and invasion of mouse mammary tumor cells 4TO7 via RhoA and Rac1 signaling pathway.
Neoplasms
N-acetylglucosaminyltransferase V modulates radiosensitivity and migration of small cell lung cancer through epithelial-mesenchymal transition.
Neoplasms
N-acetylglucosaminyltransferase V regulates extravillous trophoblast invasion through glycosylation of alpha5beta1 integrin.
Neoplasms
N-Acetylglucosaminyltransferase V regulates TGF-? response in hepatic stellate cells and the progression of steatohepatitis.
Neoplasms
N-Acetylglucosaminyltransferase V triggers overexpression of MT1-MMP and reinforces the invasive/metastatic potential of cancer cells.
Neoplasms
N-glycosylation by N-acetylglucosaminyltransferase V enhances the interaction of CD147/basigin with integrin ?1 and promotes HCC metastasis.
Neoplasms
N-Glycosylation of laminin-332 regulates its biological functions. A novel function of the bisecting GlcNAc.
Neoplasms
Negative expression of N-acetylglucosaminyltransferase V in oral squamous cell carcinoma correlates with poor prognosis.
Neoplasms
NMR structural characterization of substrates bound to N-acetylglucosaminyltransferase V.
Neoplasms
Oligosaccharide modification by N-acetylglucosaminyltransferase-V in macrophages are involved in pathogenesis of bleomycin-induced scleroderma.
Neoplasms
Overexpression and ?-1,6-N-acetylglucosaminylation-initiated aberrant glycosylation of TIMP-1; A "double whammy" strategy in colon cancer progression.
Neoplasms
Positive expressions of N-acetylglucosaminyltransferase-V (GnT-V) and beta1-6 branching N-linked oligosaccharides in human testicular germ cells diminish during malignant transformation and progression.
Neoplasms
Post-translational glycoprotein modifications regulate colon cancer stem cells and colon adenoma progression in Apc(min/+) mice through altered Wnt receptor signaling.
Neoplasms
Potential of N-glycan in cell adhesion and migration as either a positive or negative regulator.
Neoplasms
Predictive value of N-acetylglucosaminyltransferase-V for superficial bladder cancer recurrence.
Neoplasms
Preventing E-cadherin aberrant N-glycosylation at Asn-554 improves its critical function in gastric cancer.
Neoplasms
Prognostic significance of ?1,6-N-acetylglucosaminyltransferase V expression in patients with hepatocellular carcinoma.
Neoplasms
Quantitative analysis of an aberrant glycoform of TIMP1 from colon cancer serum by L-PHA-enrichment and SISCAPA with MRM mass spectrometry.
Neoplasms
Recognition of glycan and protein substrates by N-acetylglucosaminyltransferase-V.
Neoplasms
Regulation of homotypic cell-cell adhesion by branched N-glycosylation of N-cadherin extracellular EC2-3 domains.
Neoplasms
Regulation of the GnT-V promoter by transcription factor Ets-1 in various cancer cell lines.
Neoplasms
Relationship between metastasis-associated phenotypes and N-glycan structure of surface glycoproteins in human hepatocarcinoma cells.
Neoplasms
Sequences of the mouse N-acetylglucosaminyltransferase V (Mgat5) mRNA and an mRNA expressed by an Mgat5-deficient cell line.
Neoplasms
Specific posttranslational modification regulates early events in mammary carcinoma formation.
Neoplasms
Synthesis and evaluation of a radioiodinated trisaccharide derivative as a synthetic substrate for a sensitive N-acetylglucosaminyltransferase V radioassay.
Neoplasms
The Caenorhabditis elegans gene, gly-2, can rescue the N-acetylglucosaminyltransferase V mutation of Lec4 cells.
Neoplasms
The effect of receptor protein tyrosine phosphatase kappa on the change of cell adhesion and proliferation induced by N-acetylglucosaminyltransferase V.
Neoplasms
The role of N-acetylglucosaminyltransferases V in the malignancy of human hepatocellular carcinoma.
Neoplasms
Transcriptional regulation of the protocadherin ? cluster during Her-2 protein-induced mammary tumorigenesis results from altered N-glycan branching.
Neoplasms
Transforming growth factor beta up-regulates expression of the N-acetylglucosaminyltransferase V gene in mouse melanoma cells.
Neoplasms
True significance of N-acetylglucosaminyltransferases GnT-III, V and ?1,6 fucosyltransferase in epithelial-mesenchymal transition and cancer.
Neoplasms
Upregulated ?1-6 branch N-glycan marks early gliomagenesis but exhibited biphasic expression in the progression of astrocytic glioma.
Neoplasms
Upregulation of alpha and beta integrin subunits in metastatic macrophage-melanoma fusion hybrids.
Neoplasms
Upregulation of N-acetylglucosaminyltransferase-V by heparin-binding EGF-like growth factor induces keratinocyte proliferation and epidermal hyperplasia.
Neoplasms
[GnT-V overexpression in human hepatocarcinoma cells affects its migration and expression of cell adhesion molecules]
Neoplasms, Germ Cell and Embryonal
Positive expressions of N-acetylglucosaminyltransferase-V (GnT-V) and beta1-6 branching N-linked oligosaccharides in human testicular germ cells diminish during malignant transformation and progression.
Neuroblastoma
High expression of N-acetylglucosaminyltransferase V in favorable neuroblastomas: Involvement of its effect on apoptosis.
Neuroblastoma
Integrin-dependent neuroblastoma cell adhesion and migration on laminin is regulated by expression levels of two enzymes in the O-mannosyl-linked glycosylation pathway, PomGnT1 and GnT-Vb.
Neuroblastoma
Receptor tyrosine phosphatase beta (RPTPbeta) activity and signaling are attenuated by glycosylation and subsequent cell surface galectin-1 binding.
Ovarian Neoplasms
5-AZA-dC induces epigenetic changes associated with modified glycosylation of secreted glycoproteins and increased EMT and migration in chemo-sensitive cancer cells.
Ovarian Neoplasms
High expression of N-acetylglucosaminyltransferase V in mucinous tumors of the ovary.
Parasitic Diseases
N-acetylglucosaminyltransferase V-deficiency increases susceptibility to murine malaria.
Periodontitis
The role of extracellular matrix metalloproteinase inducer glycosylation in regulating matrix metalloproteinases in periodontitis.
Pheochromocytoma
Comparison of the expression of cell surface poly-N-acetyllactosamine-type oligosaccharides in PC12 cells with those in its variant PC12D.
Pheochromocytoma
N-acetylglucosaminyltranferase VB expression enhances beta1 integrin- dependent PC12 neurite outgrowth on laminin and collagen.
Pheochromocytoma
N-acetylglucosaminyltransferase V modifies TrKA protein, regulates the receptor function.
Pre-Eclampsia
The Role of MGAT5 in Human Umbilical Vein Endothelial Cells: Possible Relevance to the Pathological Mechanism of Preeclampsia.
Prostatic Neoplasms
Evaluating the function of matriptase and N-acetylglucosaminyltransferase V in prostate cancer metastasis.
Prostatic Neoplasms
Inhibition of N-acetylglucosaminyltransferase V enhances sensitivity of radiotherapy in human prostate cancer.
Prostatic Neoplasms
[Short hairpin RNA targeting N-acetylglucosaminyltransferase V inhibits the proliferation of prostate cancer PC-3 cell line]
Psoriasis
Genome-Wide Pathway Analysis Identifies Genetic Pathways Associated with Psoriasis.
Pulmonary Disease, Chronic Obstructive
The polymorphisms of FGFR2 and MGAT5 affect the susceptibility to COPD in the Chinese people.
Sarcoma
Regulation of N-acetylglucosaminyltransferase V activity. Kinetic comparisons of parental, Rous sarcoma virus-transformed BHK, and L-phytohemagglutinin-resistant BHK cells using synthetic substrates and an inhibitory substrate analog.
Sarcoma, Avian
Regulation of N-acetylglucosaminyltransferase V activity. Kinetic comparisons of parental, Rous sarcoma virus-transformed BHK, and L-phytohemagglutinin-resistant BHK cells using synthetic substrates and an inhibitory substrate analog.
Sarcoma, Avian
Transcriptional regulation of N-acetylglucosaminyltransferase V by the src oncogene.
Seminoma
Positive expressions of N-acetylglucosaminyltransferase-V (GnT-V) and beta1-6 branching N-linked oligosaccharides in human testicular germ cells diminish during malignant transformation and progression.
Small Cell Lung Carcinoma
N-acetylglucosaminyltransferase V modulates radiosensitivity and migration of small cell lung cancer through epithelial-mesenchymal transition.
Squamous Cell Carcinoma of Head and Neck
Negative expression of N-acetylglucosaminyltransferase V in oral squamous cell carcinoma correlates with poor prognosis.
Stomach Neoplasms
Clinical and prognostic implications of ?1, 6-N-acetylglucosaminyltransferase V in patients with gastric cancer.
Stomach Neoplasms
Diagnostic utility of glycosyltransferase mRNA expression in gastric cancer.
Stomach Neoplasms
Downregulation of the GnT-V gene inhibits metastasis and invasion of BGC823 gastric cancer cells.
Stomach Neoplasms
E-cadherin and adherens-junctions stability in gastric carcinoma: Functional implications of glycosyltransferases involving N-glycan branching biosynthesis, N-acetylglucosaminyltransferases III and V.
Stomach Neoplasms
Expression of N-acetylglucosaminyltransferase V in gastric cancer correlates with metastasis and prognosis.
Stomach Neoplasms
Prometastatic effect of N-acetylglucosaminyltransferase V is due to modification and stabilization of active matriptase by adding beta 1-6 GlcNAc branching.
Stomach Neoplasms
The implication of N-acetylglucosaminyltransferase V expression in gastric cancer.
Thyroid Neoplasms
Co-expression of matriptase and N-acetylglucosaminyltransferase V in thyroid cancer tissues--its possible role in prolonged stability in vivo by aberrant glycosylation.
Tics
Specific posttranslational modification regulates early events in mammary carcinoma formation.
Urinary Bladder Neoplasms
GnT-V promotes chemosensitivity to gemcitabine in bladder cancer cells through ?1,6 GlcNAc branch modification of human equilibrative nucleoside transporter 1.
Urinary Bladder Neoplasms
N-acetylglucosaminyltransferase V and beta1-6 branching N-linked oligosaccharides are associated with good prognosis of patients with bladder cancer.
Urinary Bladder Neoplasms
Predictive value of N-acetylglucosaminyltransferase-V for superficial bladder cancer recurrence.
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