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1.14.19.4: acyl-lipid (11-3)-desaturase

This is an abbreviated version!
For detailed information about acyl-lipid (11-3)-desaturase, go to the full flat file.

Word Map on EC 1.14.19.4

Reaction

an (11Z,14Z)-icosa-11,14-dienoyl-[glycerolipid]
+ 2 ferrocytochrome b5 +
O2
+ 2 H+ =
an (8Z,11Z,14Z)-icosa-8,11,14-trienoyl-[glycerolipid]
+ 2 ferricytochrome b5 + 2 H2O

Synonyms

AtSLD1, AtSLD2, Boofd8, Ca-SLDp, D8A, DELTA6-desaturase, DELTA8 acyl-CoA-dependent desaturase, DELTA8 desaturase, DELTA8 sphingobase desaturase, Delta8 sphingolipid desaturase, DELTA8-desaturase, DELTA8-fatty-acid desaturase, DELTA8-sphingolipid desaturase, FADS2, SLD, SlSLD, sphingolipid delta8 desaturase, sphingolipid DELTA8 LCB desaturase, sphingolipid DELTA8 long-chain base desaturase, sphingolipid DELTA8-desaturase, TpdesB

ECTree

     1 Oxidoreductases
         1.14 Acting on paired donors, with incorporation or reduction of molecular oxygen
             1.14.19 With oxidation of a pair of donors resulting in the reduction of O2 to two molecules of water
                1.14.19.4 acyl-lipid (11-3)-desaturase

General Information

General Information on EC 1.14.19.4 - acyl-lipid (11-3)-desaturase

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GENERAL INFORMATION
ORGANISM
UNIPROT
COMMENTARY hide
LITERATURE
malfunction
-
in enzyme knockout plants, changes in leaf cell structure under a chilling treatment are observed by transmission electron microscopy. The degree of chilling damage is greater in SlSLD-silenced plants than in control plants, indicating that SlSLD knockdown significantly reduces the chilling resistance of tomato. Compared with control plants, SlSLD-silenced plants show higher relative electrolytic leakage and malondialdehyde content, and lower superoxide dismutase and peroxidase activities after a chilling treatment. Chilling severely damages the chloroplasts in SlSLD-silenced plants, resulting in the disruption of chloroplast membranes, swelling of thylakoids, and reduced granal stacking
metabolism
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SlSLD1 and 2 are important chilling stress-responsive genes involved in sphingolipid metabolism
physiological function