1.13.99.1: inositol oxygenase
This is an abbreviated version!
For detailed information about inositol oxygenase, go to the full flat file.
Word Map on EC 1.13.99.1
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1.13.99.1
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d-glucuronate
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glucaric
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diiron
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ambience
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mixed-valent
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four-electron
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glucuronate-xylulose
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medicine
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diagnostics
- 1.13.99.1
- d-glucuronate
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glucaric
-
diiron
-
ambience
-
mixed-valent
-
four-electron
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glucuronate-xylulose
- medicine
- diagnostics
Reaction
Synonyms
amyoinositol oxygenase, AtMIOX, EC 1.13.1.11, EC 1.99.2.6, GsMIOX1a, Inositol oxygenase, inositol oxygenase 1, InOx, Kidney-specific protein 32, meso-Inositol oxygenase, MIOX, MIOX1, MIOX2, MIOX4, MIOX5, mMIOX, MOO, Myo-inositol oxygenase, OsMIOX, Oxygenase, inositol, ppMIOX, Renal-specific oxidoreductase, renal-specific oxidoreductase/myo-inositol oxygenase, RSOR/MIOX
ECTree
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Activating Compound
Activating Compound on EC 1.13.99.1 - inositol oxygenase
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D-cysteine
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best activation system: 1 mM Fe(II) and 2 mM cysteine, L-cysteine can be replaced by D-cysteine, DL-penicillamine or gamma-L-glutamyl-L-cysteine
DL-Penicillamine
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best activation system: 1 mM Fe(II) and 2 mM cysteine, L-cysteine can be replaced by D-cysteine, DL-penicillamine or gamma-L-glutamyl-L-cysteine
gamma-L-glutamyl-L-cysteine
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best activation system: 1 mM Fe(II) and 2 mM cysteine, L-cysteine can be replaced by D-cysteine, DL-penicillamine or gamma-L-glutamyl-L-cysteine
palmitate
concomitant treatment with palmitate/bovine serum albumin and activators of PKA (forskolin), PDK/PI3K (insulin), and PKC (TPA) increase Miox activity. Concomitant treatment with respective inhibitors, i.e. H89 (PKA), wortmannin (PI3K), and calphostin (PKC), reduces the activity below the levels induced by palmitate/bovine serum albumin alone, confirming that fatty acid-induced activity is phosphorylation-dependent
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1 mM Fe(II) + 4 mM quinolinate activates to 70% of the Fe(II)/cysteine system, Fe(II) alone causes very little activation, quinolinate gives considerable activation in absence of Fe(II), activation by Fe(II) and quinolinate is very temperature dependent
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increased secretion in a dose-dependent manner after high-glucose treatment
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