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Literature summary for 2.7.13.3 extracted from

  • Cerveny, J.; Sinetova, M.A.; Zavrel, T.; Los, D.A.
    Mechanisms of high temperature resistance of Synechocystis sp. PCC 6803: an impact of histidine kinase 34 (2015), Life, 5, 676-699.
    View publication on PubMedView publication on EuropePMC

Organism

Organism UniProt Comment Textmining
Synechocystis sp. P73276 GT-L
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Source Tissue

Source Tissue Comment Organism Textmining
cell culture optimal growth temperature for Synechocystis is 32-38°C, for both wild-type and DELTAHik34 mutant strains showing the same growth rates Synechocystis sp.
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Synonyms

Synonyms Comment Organism
Hik34
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Synechocystis sp.
histidine kinase 34
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Synechocystis sp.

General Information

General Information Comment Organism
malfunction DeltaHik34 mutant cells are resistant to heat stress within its first hours, they cannot recover after 24 h long high temperature treatment, while the wild-type cell population is able to recover after 24 h of cultivation at 44°C. The damage caused by high temperature depends on many factors, which differ in these experiments: light intensity, CO2 content, the growth stage, or the growth temperature before heat stress. In DELTAHik34 mutant, the content of all pigments starts to decrease after 6 h of heat stress. Heat stress phenotypes, overview Synechocystis sp.
physiological function the enzyme Hik34 plays an important role in changes in transcriptome, proteome, lipidome, and photosynthesis in response to short term heat stress. Hik34 affects the expression of sets of genes under salt and osmotic stress, under normal conditions and heat stress and under oxidative stress Synechocystis sp.