Any feedback?
Please rate this page
(literature.php)
(0/150)

BRENDA support

Literature summary for 2.7.1.67 extracted from

  • Yang, N.; Ma, P.; Lang, J.; Zhang, Y.; Deng, J.; Ju, X.; Zhang, G.; Jiang, C.
    Phosphatidylinositol 4-kinase IIIbeta is required for severe acute respiratory syndrome coronavirus spike-mediated cell entry (2012), J. Biol. Chem., 287, 8457-8467.
    View publication on PubMedView publication on EuropePMC

Inhibitors

Inhibitors Comment Organism Structure
LY294002 treatment inhibits SARS-CoV S-mediated entry into VeroE6 cells Chlorocebus sabaeus
Wortmannin treatment inhibits SARS-CoV S-mediated entry into VeroE6 cells Chlorocebus sabaeus

Organism

Organism UniProt Comment Textmining
Chlorocebus sabaeus
-
-
-
Homo sapiens
-
-
-

Source Tissue

Source Tissue Comment Organism Textmining
HEK-293 cell
-
Homo sapiens
-
Vero-E6 cell
-
Chlorocebus sabaeus
-

Synonyms

Synonyms Comment Organism
phosphatidylinositol 4-kinase IIIbeta
-
Homo sapiens
phosphatidylinositol 4-kinase IIIbeta
-
Chlorocebus sabaeus

General Information

General Information Comment Organism
malfunction knockdown of isozyme phosphatidylinositol 4-kinase IIIbeta inhibits SARS-CoV S-mediated entry into VeroE6 cells Chlorocebus sabaeus
physiological function isozyme phosphatidylinositol 4-kinase IIIbeta is required for severe acute respiratory syndrome coronavirus spike-mediated cell entry, overview Chlorocebus sabaeus
physiological function isozyme phosphatidylinositol 4-kinase IIIbeta is required for severe acute respiratory syndrome coronavirus spike-mediated cell entry. PI4P plays an essential role in SARS-CoV spike-mediated entry, which is regulated by the PI4P lipid microenvironment, overview Homo sapiens