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Literature summary for 2.4.1.109 extracted from

  • Snoei, J.; Urbach, H.; Engels, G.; Fassunke, J.; von Lehe, M.; Becker, A.J.; Majores, M.
    Genetic alterations of protein-o-mannosyltransferase-1 in glioneuronal and glial brain tumors with subarachnoid spread (2009), Neuropathology, 29, 116-124.
    View publication on PubMed

Organism

Organism UniProt Comment Textmining
Homo sapiens Q9Y6A1
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Source Tissue

Source Tissue Comment Organism Textmining
glioma cell of brain with subarachnoid spread. Distinct POMT1 alterations may contribute to a functional impairment of protein-omannosyltransferase activity. In individual tumor specimens selective alterations of the POMT1 gene may be compatible with clonal evolution of distinct sublocations Homo sapiens
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neuroglioma cell with subarachnoid spread. Distinct POMT1 alterations may contribute to a functional impairment of protein-O-mannosyltransferase activity. In individual tumor specimens selective alterations of the POMT1 gene may be compatible with clonal evolution of distinct sublocations Homo sapiens
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Synonyms

Synonyms Comment Organism
POMT1
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Homo sapiens
protein-O-mannosyltransferase-1
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Homo sapiens