Literature summary for 1.6.5.10 extracted from

Zai, C.C.; Tiwari, A.K.; Basile, V.; de Luca, V.; Mueller, D.J.; Voineskos, A.N.; Remington, G.; Meltzer, H.Y.; Lieberman, J.A.; Potkin, S.G.; Kennedy, J.L.
Oxidative stress in tardive dyskinesia: Genetic association study and meta-analysis of NADPH quinine oxidoreductase 1 (NQO1) and superoxide dismutase 2 (SOD2, MnSOD) genes (2009), Prog. Neuropsychopharmacol. Biol. Psychiatry, 34, 50-56.

Search for more literature for 1.6.5.10

Application

Application	Comment	Organism
medicine	Pro187Ser polymorphism in NQO1 has a limited role in the development of Tardive dyskinesia (a potentially irreversible side effect of antipsychotic medication treatment that occurs in approximately 25% of chronically treated schizophrenia patients)	Homo sapiens

Protein Variants

Protein Variants	Comment	Organism
P187S	Pro187Ser polymorphism in NQO1 has a limited role in the development of Tardive dyskinesia (a potentially irreversible side effect of antipsychotic medication treatment that occurs in approximately 25% of chronically treated schizophrenia patients)	Homo sapiens

Organism

Organism	UniProt	Comment	Textmining
Homo sapiens	-	-	-

Synonyms

Synonyms	Comment	Organism
NADPH quinone oxidoreductase 1	-	Homo sapiens
NQO1	-	Homo sapiens

Use of this online version of BRENDA is free under the CC BY 4.0 license. See terms of use for full details.

Release 2023.1 (January 2023)