Application | Comment | Organism |
---|---|---|
medicine | in rabbits with heart failure induced by myocardial infarction, treatment with inhibitor apocynin reduces NADPH oxidase activity, subunit p47phox protein, oxidative stress, myocyte apoptosis, and Bax protein, increases Bcl-2 protein, and ameliorates left ventricular dilatation and dysfunction | Oryctolagus cuniculus |
Inhibitors | Comment | Organism | Structure |
---|---|---|---|
apocynin | in rabbits with heart failure induced by myocardial infarction, apocynin reduces NADPH oxidase activity, subunit p47phox protein, oxidative stress, myocyte apoptosis, and Bax protein, increases Bcl-2 protein, and ameliorates left ventricular dilatation and dysfunction | Oryctolagus cuniculus |
Organism | UniProt | Comment | Textmining |
---|---|---|---|
Oryctolagus cuniculus | - |
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Source Tissue | Comment | Organism | Textmining |
---|---|---|---|
heart | rabbits with heart failure induced by myocardial infarction exhibit left ventricular dilatation and systolic dysfunction. Changes are associated with increases in NADPH oxidase activity, subunit p47phox protein expression, 8-hydroxydeoxyguanosine expression, 4-hydroxy-2-nonenal expression, myocyte apoptosis, and Bax protein and a decrease in Bcl-2 protein | Oryctolagus cuniculus | - |