Localization | Comment | Organism | GeneOntology No. | Textmining |
---|---|---|---|---|
mitochondrion | - |
Homo sapiens | 5739 | - |
Natural Substrates | Organism | Comment (Nat. Sub.) | Natural Products | Comment (Nat. Pro.) | Rev. | Reac. |
---|---|---|---|---|---|---|
additional information | Homo sapiens | enzyme deficiency may ba a cause of autism, physiological alterations in case of enzyme deficiency are in TCA energy metabolism, ammonia detoxification, and synthesis of omega-3 DHA, and elevated acyl-carnitine levels, the mitochondrial enzyme is responsible for beta-oxidation of branched and unsaturated fatty acids with preference for long-chain fatty acids | ? | - |
? |
Organism | UniProt | Comment | Textmining |
---|---|---|---|
Homo sapiens | - |
8-year-old male autistic patient | - |
Substrates | Comment Substrates | Organism | Products | Comment (Products) | Rev. | Reac. |
---|---|---|---|---|---|---|
linoleoyl-CoA + acceptor | - |
Homo sapiens | 2,9,12-octadecatrienoyl-CoA + reduced acceptor | - |
? | |
additional information | enzyme deficiency may ba a cause of autism, physiological alterations in case of enzyme deficiency are in TCA energy metabolism, ammonia detoxification, and synthesis of omega-3 DHA, and elevated acyl-carnitine levels, the mitochondrial enzyme is responsible for beta-oxidation of branched and unsaturated fatty acids with preference for long-chain fatty acids | Homo sapiens | ? | - |
? | |
additional information | enzyme has broad substrate specificity with preference for medium- and long-chain fatty acids, overview | Homo sapiens | ? | - |
? | |
oleoyl-CoA + acceptor | - |
Homo sapiens | 2,9-octadecadienoyl-CoA + reduced acceptor | - |
? |
Synonyms | Comment | Organism |
---|---|---|
LCAD | - |
Homo sapiens |
long chain acyl-CoA dehydrogenase | - |
Homo sapiens |