2.6.1.76: diaminobutyrate-2-oxoglutarate transaminase
This is an abbreviated version!
For detailed information about diaminobutyrate-2-oxoglutarate transaminase, go to the full flat file.
Word Map on EC 2.6.1.76
-
2.6.1.76
-
medicine
-
1,3-diaminopropane
-
ectoine
-
halophilic
-
methanotrophs
-
halotolerant
-
methanol
-
acinetobacter
-
osmoadaptation
-
alcaliphilum
-
baumannii
-
aminotransferases
-
synthesis
- 2.6.1.76
- medicine
- 1,3-diaminopropane
- ectoine
-
halophilic
-
methanotrophs
-
halotolerant
- methanol
- acinetobacter
-
osmoadaptation
-
alcaliphilum
- baumannii
- aminotransferases
- synthesis
Reaction
Synonyms
(Pl)EctB, 2,4-diaminobutyrate 4-aminotransferase, 2,4-diaminobutyrate aminotransferase, DABA aminotransferase, DABA AT, DABA-AT, DABA:2-KG 4 aminotransferase, Diaminobutyrate transaminase, EctB, L-2,4-diaminobutyrate:2-ketoglutarate 4-aminotransferase, L-diaminobutyric acid transaminase, More
ECTree
Advanced search results
Application
Application on EC 2.6.1.76 - diaminobutyrate-2-oxoglutarate transaminase
Please wait a moment until all data is loaded. This message will disappear when all data is loaded.
medicine
synthesis
medicine
-
responsible for many human infections including otitis media, meningitis, epiglottis and pneumonia
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
medicine
-
drug development, Acinetobacter sp. can be part of the normal flora of human skin but also of opportunistic infections like septicemia, pneumonia, endocarditis, meningitis, skin and wound sepsis or urinary tract infections, clinically important isolates show widespread and increasing resistance to many commonly used antibiotics
-
production of ectoine in Escherichia coli. The Escherichia coli regulatory protein AraC is engineered to recognize ectoine as it snon-natural effector and to activate transcription upon ectoine binding. The ectoine biosynthetic cluster from Halomonas elongata is cloned into Escherichia coli. By engineering the rate-limiting enzyme L-2,4-diaminobutyric acid aminotransferase EctB, ectoine production and the specific activityof the EctB mutant are increased
synthesis
-
production of ectoine in Escherichia coli. The Escherichia coli regulatory protein AraC is engineered to recognize ectoine as it snon-natural effector and to activate transcription upon ectoine binding. The ectoine biosynthetic cluster from Halomonas elongata is cloned into Escherichia coli. By engineering the rate-limiting enzyme L-2,4-diaminobutyric acid aminotransferase EctB, ectoine production and the specific activityof the EctB mutant are increased
-