2.4.1.34: 1,3-beta-glucan synthase
This is an abbreviated version!
For detailed information about 1,3-beta-glucan synthase, go to the full flat file.
Word Map on EC 2.4.1.34
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2.4.1.34
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candida
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echinocandins
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albicans
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caspofungin
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cytokinesis
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micafungin
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amphotericin
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candidiasis
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rho1
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anidulafungin
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powdery
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plasmodesmata
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mildew
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pneumocandins
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microspore
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papulacandins
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calcofluor
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symplastic
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candidemia
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aculeacin
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echinocandin-resistant
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agriculture
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analysis
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medicine
- 2.4.1.34
- candida
-
echinocandins
- albicans
- caspofungin
-
cytokinesis
- micafungin
- amphotericin
- candidiasis
- rho1
- anidulafungin
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powdery
-
plasmodesmata
- mildew
- pneumocandins
- microspore
- papulacandins
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calcofluor
- symplastic
-
candidemia
-
aculeacin
-
echinocandin-resistant
- agriculture
- analysis
- medicine
Reaction
Synonyms
(1,3)-beta-D-glucan (curdlan) synthase, (1,3)-beta-D-glucan synthase, (1,3)-beta-glucan (callose) synthase, (1,3)beta-D-glucan synthase, 1,3-beta-D-glucan 3-beta-D-glucosyltransferase, 1,3-beta-D-glucan synthase, 1,3-beta-D-glucan synthetase, 1,3-beta-D-glucan-UDP glucosyltransferase, 1,3-beta-glucan synthase, 1,3-beta-glucan synthase (GS), 1,3-beta-glucan-uridine diphosphoglucosyltransferase, Arabidopsis beta-1,3-glucan synthase, beta(1,3)-D-glucan synthase, beta(1,3)glucan synthase, beta-1,3-glucan synthase, beta-1,3-glucan synthetase, beta-glucan synthase, Bgs1p, bgs2p, Bgs3p, Bgs4p, callose synthase, callose synthetase, CalS, CrdS, curdlan synthase, Fks, FKS-1, Fks1p, FKS2, GCS1, GLS1, glucan synthase (GS), glucan synthase-like, glucan synthase-like 1, GS-II, Gsc-1, Gsc1, GSC2, GSL, GSL1, GSL8, MaFKS, More, paramylon synthetase, PbFKS1, PMR, PNEG_03180, PNEJI1_001061, UDP-glucose-1,3-beta-glucan glucosyltransferase, UDP-glucose-beta-glucan glucosyltransferase, UDP-glucose:(1,3)beta-glucan synthase, UDPglucose-1,3-beta-D-glucan glucosyltransferase, uridine diphosphoglucose-1,3-beta-glucan glucosyltransferase, uridine-diphosphate glucose:1,3-beta-D-glucosyltransferase, ZT1E4_G10604
ECTree
2.4.1.34 1,3-beta-glucan synthaseAdvanced search results
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results (Engineering
Engineering on EC 2.4.1.34 - 1,3-beta-glucan synthase
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PROTEIN VARIANTS 
ORGANISM
UNIPROT
COMMENTARY 
LITERATURE
S715P
mutation increases complementation efficiency of a Saccharomyces cerevisiae Gsc1 mutant and corresponds to a decrease of sensitivity to caspofungin
S718P
mutation increases complementation efficiency of a Saccharomyces cerevisiae Gsc1 mutant and corresponds to a decrease of sensitivity to caspofungin
S718P/F714S
mutation further increases complementation efficiency of a Saccharomyces cerevisiae Gsc1 mutant and corresponds to a decrease of sensitivity to caspofungin
S718P
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mutation increases complementation efficiency of a Saccharomyces cerevisiae Gsc1 mutant and corresponds to a decrease of sensitivity to caspofungin
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S718P/F714S
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mutation further increases complementation efficiency of a Saccharomyces cerevisiae Gsc1 mutant and corresponds to a decrease of sensitivity to caspofungin
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S719P
additional information
S719P
mutation increases complementation efficiency of a Saccharomyces cerevisiae Gsc1 mutant and corresponds to a decrease of sensitivity to caspofungin
S719P
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mutation increases complementation efficiency of a Saccharomyces cerevisiae Gsc1 mutant and corresponds to a decrease of sensitivity to caspofungin
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additional information
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knockout mutants of CalS5 gene result in severe reduction in fertility by degeneration of microspores. Callose deposition in mutants is nearly completely lacking and pollen exine wall is not formed properly
additional information
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silencing of enzyme genes GSL5, GSL6, GSL11 with RNAi: both wound callose and papillary callose are absent in lines transformed with GSL5 dsRNAi, but unaffected in GSL6 and GSL11 RNAi lines. Absence of callose in palpillae or haustorial complexes correlates with effective growth cessation of several normally virulent powdery mildew species and of Peronospora parasitica
additional information
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mutations in GSL8 produce seedlings with pleiotropic defects during embryogenesis and early vegetative growth, with cell wall stubs, two nuclei in one cell, and other defects in cell division in homozygous gsl8 insertional alleles. In addition, gsl8 mutants and inducible RNA interference lines of GSL8 show reduced callose deposition at cell plates and/or new cell walls, phenotypes, overview
additional information
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isolation of ten different temperature-sensitive fks1 mutants after random mutagenesis, quantitative cell morphology and biochemical properties of fks1 mutants, the putative catalytic subunit of the enzyme, the mutants show altered cell wall components and defects in endocytosis, pleiotropic phenotypes, overview. Some fks-1 mutants show intragenic complementation between fks1 mutations, while others do not
additional information
gene deletion strain, no viable spores, bgs4+ shut-off produces cell lysis at growing poles and mainly at the septum prior to cytokinesis
additional information
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gene deletion strain, no viable spores, bgs4+ shut-off produces cell lysis at growing poles and mainly at the septum prior to cytokinesis
additional information
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in the absence of Bgs1p abnormal septa are formed, but the cells cannot separate and eventually die, mutant phenotype, overview
additional information
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null bgs2 diploid mutants form four spore-like compartments inside each ascus, in which the electron-lucent layer is thinner and darker than in the wild-type, and the spores are unviable
additional information
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inhibition profile of wild-type and mutant enzymes with antifungal drugs, overview
