1.7.1.7: GMP reductase
This is an abbreviated version!
For detailed information about GMP reductase, go to the full flat file.
Word Map on EC 1.7.1.7
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1.7.1.7
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purine
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imp
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salvage
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hypoxanthine
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impdhs
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hypoxanthine-guanine
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adenylosuccinate
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medicine
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drug development
- 1.7.1.7
- purine
- imp
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salvage
- hypoxanthine
- impdhs
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hypoxanthine-guanine
- adenylosuccinate
- medicine
- drug development
Reaction
Synonyms
EC 1.6.6.8, GMPR, GMPR-I, GMPR-II, GMPR2, guaC, Guanosine 5'-monophosphate oxidoreductase, guanosine 5'-monophosphate reductase, guanosine monophosphate reductase, guanosine monophosphate reductase 2, guanosine-5'-monophosphate reductase, guanylate reductase, HsGMPR1, HsGMPR2, LmGMPR, Lmj17.0725, LMJF_17_0725, NADPH:GMP oxidoreductase (deaminating), reductase, guanylate, Tb927.5.2080, TbGMPR, TcGMPR, TCIL3000_5_1940
ECTree
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Disease
Disease on EC 1.7.1.7 - GMP reductase
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Alzheimer Disease
Guanosine monophosphate reductase 1 is a potential therapeutic target for Alzheimer's disease.
Breast Neoplasms
Lack of expression of the proteins GMPR2 and PPAR? are associated with the basal phenotype and patient outcome in breast cancer.
Carcinogenesis
Lack of expression of the proteins GMPR2 and PPAR? are associated with the basal phenotype and patient outcome in breast cancer.
Leukemia
Cloning and functional characterization of GMPR2, a novel human guanosine monophosphate reductase, which promotes the monocytic differentiation of HL-60 leukemia cells.
Leukemia
Reciprocal alterations of GMP reductase and IMP dehydrogenase activities during differentiation in HL-60 leukemia cells.
Melanoma
A purine nucleotide biosynthesis enzyme guanosine monophosphate reductase is a suppressor of melanoma invasion.
Neoplasms
Characterizing and optimizing human anticancer drug targets based on topological properties in the context of biological pathways.
Neoplasms
Cloning and functional characterization of GMPR2, a novel human guanosine monophosphate reductase, which promotes the monocytic differentiation of HL-60 leukemia cells.
Starvation
Inhibition of cellular growth by increased guanine nucleotide pools. Characterization of an Escherichia coli mutant with a guanosine kinase that is insensitive to feedback inhibition by GTP.
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