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EXPRESSION
ORGANISM
UNIPROT
LITERATURE
BCKD and BCKDK mRNA decrease significantly immediately after CCI-induced traumatic brain injury (TBI) in the rat. Brain BCKD mRNA levels decrease significantly in the craniotomy, contralateral CCI-induced TBI, and ipsilateral CCI at 4 and 24 h post-TBI, and the decrease remains significant in the contralateral CCI-induced TBI 3 days post-CCI. Phosphorylated BCKD proteins (pBCKD) increase significantly in the ipsilateral-CCI-induced TBI hemisphere. Glutamate treatment (0.025 mM for 24 h) significantly decreases pBCKD protein in neurons
BCKD and BCKDK mRNA decrease significantly immediately after CCI-induced traumatic brain injury (TBI) in the rat. Brain BCKD mRNA levels decrease significantly in the craniotomy, contralateral CCI-induced TBI, and ipsilateral CCI at 4 and 24 h post-TBI, and the decrease remains significant in the contralateral CCI-induced TBI 3 days post-CCI. Phosphorylated BCKD proteins (pBCKD) increase significantly in the ipsilateral-CCI-induced TBI hemisphere. Glutamate treatment (0.025 mM for 24 h) significantly decreases pBCKD protein in neurons
BCKD and BCKDK mRNA decrease significantly immediately after CCI-induced traumatic brain injury (TBI) in the rat. Brain BCKD mRNA levels decrease significantly in the craniotomy, contralateral CCI-induced TBI, and ipsilateral CCI at 4 and 24 h post-TBI, and the decrease remains significant in the contralateral CCI-induced TBI 3 days post-CCI. Phosphorylated BCKD proteins (pBCKD) increase significantly in the ipsilateral-CCI-induced TBI hemisphere. Glutamate treatment (0.025 mM for 24 h) significantly decreases pBCKD protein in neurons